Stealth Adapted Coronaviruses Resulting from the Use of Covid-19 Vaccines

Abstract

The continuing emergence of variant forms of the SARS-CoV-2 virus is the probable\n consequence of using the current Covid-19 vaccines. These vaccines do not induce the\n same immunity as do naturally occurring infections.First, the vaccines are given by\n intramuscular injections. This is far less effective than natural infection in stimulating\n the development of virus specific immunoglobulin A (IgA) producing cells and cytotoxic\n T cells (CTL) within the respiratory mucosa. Virus exposure in a previously vaccinated\n individual with limited mucosal immunity increases the risk of a persistent, subclinical\n infection, which will initially be restricted to the superficial mucosa. NIH and CDC\n officials have alluded to this possibility in advising those who have been vaccinated\n to continue wearing masks lest they may infect others. The second major distinction\n between the Covid-19 vaccines and natural infection is the FDA allowance of using\n only one component as the antigen, namely the spike protein. Deletion or other modifications\n of a single targeted component can occur more readily as an immune evasion mechanism\n than concurrent genetic modifications of multiple antigenic components. Covid-19 vaccine\n evoked immunity will, therefore, exert a strong immunoselective pressure for major\n modifications or deletion of the spike protein. With successive additional changes\n in the few remaining viral components that are normally targeted by cellular immunity,\n as well as the incorporation of sufficient genetic sequences from cells and other\n microbes; non-immunogenic, pathogenic viruses will then emerge. These viruses will\n no longer be immunologically restricted to the respiratory mucosa and will become\n more widespread within the body. The immune evasion/escape mechanism utilized in this\n manner is termed stealth adaptation. It was initially identified in the cytomegaloviruses\n of monkeys used to produce polio vaccines. Not only were these viruses probably involved\n in causing AIDS, but they can account for the rise in many chronic illnesses, such\n as autism and the chronic fatigue syndrome (CFS). Until proven otherwise, the neuropsychiatric\n symptoms of the Long Covid syndrome in previously healthy individuals, are consistent\n with brain infection with stealth adapted coronaviruses. This illness is, therefore,\n likely to be infectious, including the possibility of transplacental transmission.\n Testing for stealth adapted viruses in these patients is best performed using virus\n cultures followed by genetic sequencing. Even though cellular immunity fails to effectively\n suppress stealth adapted viruses, these viruses as well as the conventional viruses\n from which they are derived, are still susceptible to a non-immunological anti-virus\n defense mechanism mediated by the alternative cellular energy (ACE) pathway. This\n pathway is reflected in an added kinetic activity of the body’s fluids.The environmental\n life-force energy for the ACE pathway is called KELEA (Kinetic Energy Limiting Electrostatic\n Attraction). Water with high levels of KELEA is available for clinical studies.Enhancing\n the ACE pathway in those who are susceptible to severe Covid-19 illness and in Long\n Covid syndrome patients is arguably preferable to risking the development of new forms\n of stealth adapted viruses by using the current Covid-19 vaccines.