Akashdeep Singh
Christian Medical College & Hospital
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Publication
Featured researches published by Akashdeep Singh.
Mycoses | 2010
Alok Nath; Ritesh Agarwal; Akashdeep Singh; Ruchi Gupta; Kusum Joshi
Dysphonia in patients with bronchial asthma is generally ascribed to vocal‐cord abnormalities or steroid myopathy secondary to inhaled corticosteroids. Herein, we report the case of a 55‐year‐old male patient – a diagnosed case of bronchial asthma being on inhaled corticosteroids – who presented with dysphonia and was diagnosed to be suffering from Aspergillus laryngotracheobronchitis.
Lung India | 2012
Akashdeep Singh; Rupinder Kaur
Acute lupus pneumonitis is an uncommon but life threatening condition associated with systemic lupus erythematosus. We report the case of a young female who presented to us with acute hypoxemic respiratory failure secondary to acute lupus pneumonitis as initial presenting manifestation of lupus. She was managed with non-invasive ventilation and pulse steroids, with which she had dramatic improvement.
Lung India | 2013
Akashdeep Singh; Parminder Singh; Us Sidhu
We describe the case summary of a 70-year-old man diagnosed with interstitial lung disease due to prolonged nitrofurantoin therapy. Despite honeycombing confirmed by computed tomography of the thorax, symptoms and radiographic findings disappeared within 1 month after withdrawal of nitrofurantoin. The case highlights the fact that nitrofurantoin-induced lung disease may run a benign course and respond favorably despite radiographic evidence of established lung fibrosis (honey combing).
Lung India | 2016
Akashdeep Singh; Deepinder Chhina; Rk Soni; Chandan Kakkar; Us Sidhu
Background: Pulmonary nocardiosis is a rare but a life-threatening infection caused by Nocardia spp. The diagnosis is often missed and delayed resulting in delay in appropriate treatment and thus higher mortality. Aim: In this study, we aim to evaluate the clinical spectrum and outcome of patients with pulmonary nocardiosis. Methods: A retrospective, 5-year (2009-2014) review of demographic profile, risk factors, clinical manifestations, imaging findings, treatment, and outcome of patients with pulmonary nocardiosis admitted to a tertiary care hospital. Results: The median age of the study subjects was 54 years (range, 16-76) and majority of them (75%) were males. The risk factors for pulmonary nocardiosis identified in our study were long-term steroid use (55.6%), chronic lung disease (52.8%), diabetes (27.8%), and solid-organ transplantation (22.2%). All the patients were symptomatic, and the most common symptoms were cough (91.7%), fever (78%), and expectoration (72%). Almost two-third of the patients were initially misdiagnosed and the alternative diagnosis included pulmonary tuberculosis (n = 7), community-acquired pneumonia (n = 5), lung abscess (n = 4), invasive fungal infection (n = 3), lung cancer (n = 2), and Wegener′s granulomatosis (n = 2). The most common radiographic features were consolidation (77.8%) and nodules (56%). The mortality rate for indoor patients was 33% despite treatment. Higher mortality rate was observed among those who had brain abscess (100.0%), HIV positivity (100%), need for mechanical ventilation (87.5%), solid-organ transplantation (50%), and elderly (age > 60 years) patients (43%). Conclusion: The diagnosis of pulmonary nocardiosis is often missed and delayed resulting in delay in appropriate treatment and thus high mortality. A lower threshold for diagnosing pulmonary nocardiosis needs to be exercised, in chest symptomatic patients with underlying chronic lung diseases or systemic immunosuppression, for the early diagnosis, and treatment of this uncommon but potentially lethal disease. Despite treatment mortality remains high, especially in those with brain abscess, HIV positivity, need for mechanical ventilation, solid-organ transplantation, and elderly.
The American Journal of Medicine | 2013
Jaspreet Singh; Akashdeep Singh
In the physical findings of “Cyanosis,” presented by McMullen and Patrick, there is no mention of differential cyanosis, which is an important bedside finding. Differential cyanosis refers to the appearance of cyanosis in both lower extremities with a pink right upper extremity. This is seen in patent ductus arteriosus with pulmonary arterial hypertension. The deoxygenated blood in the pulmonary artery goes through the patent ductus arteriosus, empties into the aorta, and flows to the lower half of the body. The upper half of the body continues to get oxygenated blood from the left ventricle. If the patent ductus enters the aorta below the left subclavian artery, the left arm is pink; if it enters above the left subclavian artery, the left arm is blue. Differential cyanosis in a neonate indicates persistent pulmonary hypertension of the newborn and left-heart abnormalities (aortic arch hypoplasia, interrupted aortic arch, critical coarctation, and critical aortic stenosis).
Lung India | 2013
Akashdeep Singh; Gupreet Singh Wander
The diagnosis of pneumonia is clinical, based on the history of lower respiratory tract symptoms, physical, and/or radiographic signs of consolidation. Several diseases such as congestive heart failure, pulmonary embolism, and chemical pneumonitis may present with similar symptoms, signs, and chest radiographs, thus delaying the definitive diagnosis and initiation of appropriate treatment. Unilateral pulmonary edema (UPE) is a rare clinical entity that is often misdiagnosed at first as a focal lung disease. We have presented an unusual case of left-sided UPE in a 76-year-old man who developed acute heart failure resulting from dietary and pharmacological noncompliance. The patient was successfully managed with decongestive therapy and non-invasive mechanical ventilation.
Intercom - Revista Brasileira de Ciências da Comunicação | 2013
Robert James; Akashdeep Singh; Rupinder Kaur
Physical and neurologic examinations were normal. Chest radiography revealed a partially calcified mass in the right upper zone area [Figure 1]. Routine hematological investigations and repeated bacteriological examinations Address for correspondence: Dr. Akash Deep Singh, Department of Pulmonary Medicine, Christian Medical College and Hospital, Ludhiana-141008, Punjab, India. E-mail: [email protected]
Canadian Journal of Emergency Medicine | 2008
Akashdeep Singh
May • mai 2008; 10 (3) CJEM • JCMU 197 role of alfentanil with or without propofol in patients undergoing PSA and found no difference in the propofol dose, time of procedure, changes from baseline end-tidal CO2 or hypoxia. However, there were more supportive airway measures required in the patients who received alfentanil (34.1% alfentanil/propofol v. 12.8% propofol alone, p = 0.02). Clearly this area requires further study, not only to determine which opioid analgesic is the optimal agent, but also whether routine analgesia is necessary at all in the setting of PSA performed with propofol.
Clinical Infectious Diseases | 2007
Akashdeep Singh
Annals of Internal Medicine | 2008
Akashdeep Singh
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Post Graduate Institute of Medical Education and Research
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