C. Anne Wallen

Evidence for an unconventional radiosensitivity of rat 9L subcutaneous tumors.

The cellular radiosensitivity of rat 9L subcutaneous (sc) tumors was determined by measuring cell survival with an in vivo to in vitro colony-forming assay. The radiosensitivity of 9L tumor cells did not vary with changes in tumor weight (0.03-2.0 g), appearance of necrosis, or the use of anesthesia. None of the survival curves for cells from air-breathing rats had the traditional break normally associated with the presence of a hypoxic fraction. However, the composite survival curve showed a sensitivity (

The Combined Effects of Shock Waves and Cisplatin Therapy on Rat Prostate Tumors

D_{0}=3.6

Relationship between DNA damage, DNA repair, metabolic state and cell lethality

Gy) intermediate between and statistically different from that observed either for exponential cells from monolayer cultures (

Biodistribution of misonidazole and 1,3-bis(2-chloroethyl)1-nitrosourea (BCNU) in rats bearing unclamped and clamped 9L subcutaneous tumors☆

D_{0}=1.8

Feasibility of estimating the temperature distribution in a tumor heated by a waveguide applicator

Gy) or single cells from dissociated tumors (

Relationship of cell survival, drug dose, and drug uptake after 1-O-octadecyl-2-O-methyl-rac-glycero-3-phosphocholine treatment

D_{0}=2.1

Characterization of nuclear matrices prepared without salt extraction

Gy), and tumor cells from

Influence of Location within a Tumor on the Survival of Dissociated Cells Heated in Vitro

{\rm N}_{2}\text{-asphyxiated}

Radiation-Induced DNA Damage in Tumors and Normal Tissues. VI. Estimation of the HypoxicFraction of Experimental Tumors1

rats (

Effect of administration schedules on the potentiation of 1,3-bis(2-chloroethyl)-1-nitrosourea (BCNU) by misonidazole in subcutaneous 9l tumors

D_{0}=5.3