Network


Latest external collaboration on country level. Dive into details by clicking on the dots.

Hotspot


Dive into the research topics where David A. Cottrell is active.

Publication


Featured researches published by David A. Cottrell.


American Journal of Orthodontics and Dentofacial Orthopedics | 1996

Diagnosis of macroglossia and indications for reduction glossectomy

Larry M. Wolford; David A. Cottrell

Macroglossia can create dentomusculoskeletal deformities, instability of orthodontic and orthognathic surgical procedures, and masticatory, speech, and airway problems. The cause of macroglossia must be clearly defined, and true macroglossia separated from pseudomacroglossia (displacement of the tongue created by anatomic factors other than tongue size alone). This article discusses the signs and symptoms of macroglossia, including the clinical and radiographic features, treatment techniques, as well as previously reported results. Cases are shown to illustrate the applicability of this technique.


Journal of Oral and Maxillofacial Surgery | 1998

Long-term evaluation of the use of coralline hydroxyapatite in orthognathic surgery.

David A. Cottrell; Larry M. Wolford

PURPOSE This prospective study was designed to evaluate the long-term clinical and radiographic results of porous block hydroxyapatite (PBHA) used as a synthetic bone graft in orthognathic surgery and craniofacial augmentation. PATIENTS AND MATERIALS A total of 245 consecutive patients were treated initially. Inclusion criteria for this study included a minimum clinical and radiographic follow-up of 5 years. In addition, all patients with known implant failures were included regardless of whether they met the study criteria. There were 111 patients that met the criteria for inclusion in this study. All patients had undergone orthognathic surgery with rigid fixation and had had inlay or onlay PBHA implants placed. Ninety-six percent of the implants were placed through an intraoral approach. Long-term postoperative radiographs were visually compared with immediate postoperative radiographs for implant position, stability, resorption, and other significant radiographic changes. The clinical examination evaluated for signs and symptoms of infection, wound dehiscence, implant exposure, implant displacement, changes in the overlying mucosa, and development of oronasal or oroantral fistulae. RESULTS Four hundred seventy-one implants were placed: 403 in the maxilla, 44 in the mandible, and 24 in the periorbital region. There were 289 implants placed in direct communication with the maxillary sinus. The average follow-up time was 7.2 years (range, 5.0 to 10.3 years). Twenty-three implants (4.9%) were removed during the evaluation period. Lateral maxillary wall grafting had 95.7% success, with nine implants being lost in three patients. One chin implant was removed because of dissatisfaction with the aesthetics. Seven (14%) midpalatal implants used for maxillary expansion were lost, primarily because of exposure of the implant to the oral or nasal cavity at the time of surgery. When PBHA was used for alveolar cleft grafting, there was a 100% failure rate. CONCLUSION The use of PBHA as a bone graft substitute in orthognathic surgery and for facial augmentation showed a high percentage of success and efficacy. However, adequate soft tissue coverage in the nasal floor and on the palate are paramount for success of midpalatal implants. PBHA should not be used for alveolar cleft grafting. Rigid fixation for inlay implants in the maxilla is important to provide stress shielding of the material and minimize micromovement during the initial healing phase.


Journal of Oral and Maxillofacial Surgery | 1994

Altered orthognathic surgical sequencing and a modified approach to model surgery

David A. Cottrell; Larry M. Wolford

Advances in orthognathic surgical treatment planning and in techniques for complex, simultaneous maxillary and mandibular repositioning have resulted in improved surgical accuracy. In traditional surgical sequencing, maxillary surgery is performed first; the maxilla is set, with or without an intermediate splint, using external reference points to verify and/or determine appropriate movement. However, errors in model surgery and intermediate splint fabrication can lead to surgical inaccuracy despite good surgical technique. In repositioning the maxilla first, when thin bony walls are present, and/or in conjunction with large mandibular advancements, maxillary shifting may occur when maxillomandibular fixation is applied. Soft tissue tension and surgical manipulation in this sequencing technique may result in a less desirable functional and esthetic outcome. This article presents an alternative to surgical sequencing and a modification of model surgery techniques to improve surgical accuracy, and thus predictability and stability of the results.


Journal of Oral and Maxillofacial Surgery | 2000

Management of heparin therapy in the high-risk, chronically anticoagulated, oral surgery patient: A review and a proposed nomogram

Pushkar Mehra; David A. Cottrell; Susan C. Bestgen; Donald F. Booth

PURPOSE This study analyzes the use of a standard nomogram that can help reduce the level of anticoagulation preoperatively to effectively manage perioperative heparin therapy in chronically anticoagulated oral surgery patients who are at high risk for thromboembolism. PATIENTS AND METHODS Twenty patients with significant cardiovascular disease, ranging in age from 56 to 79 years and requiring oral surgery, were randomly divided into 2 groups. All patients were on chronic warfarin therapy, and perioperative heparinization was recommended by their cardiologist. Group A (n = 10) had their anticoagulation therapy managed with the use of a standard nomogram. The heparin therapy for group B (n = 10) was managed without the use of the nomogram. The records of all patients were analyzed for therapeutic efficacy of heparinization, number of laboratory tests required, duration of hospitalization, and complications related to heparinization. RESULTS Patients in group A did significantly better in all parameters when compared with group B patients. There were no complications in group A, whereas there was a 20% incidence of complications related to anticoagulation therapy in group B. CONCLUSIONS The use of a standard nomogram to manage anticoagulation therapy in the oral surgery patient requiring heparinization is strongly recommended. This provides optimal therapeutic benefit, decreases the incidence of complications, and makes the hospitalization less costly and more comfortable for the patient.


Journal of Oral and Maxillofacial Surgery | 1999

Life-threatening, delayed epistaxis after surgically assisted rapid palatal expansion: A case report

Pushkar Mehra; David A. Cottrell; Alfonso Caiazzo; Robert Lincoln

Lutcavage GJ: Traumatic facial artery aneurysm and arteriovenous fistula: Case report. J Oral Maxillofac Surg 50:402, 1992 Wineland PL, Topazian RG, Marble HB Jr: False aneurysm of the facial artery. J Oral Surg 34:642, 1976 Akker HP van den, Lijn F van der: A false aneurysm of the facial artery as a complication of circumferential wiring. Oral Surg Oral Med Oral Path01 37:514,1974 Schwartz SH, Blankenship BJ, Stout RA: False aneurysm of the facial artery: Report of case. J Oral Surg 29:672,1971 Bresner M, Brekke J, Dubit J, et al: False aneurysm of the facial region. J Oral Surg 30:307, 1972 Cohen SM: Vascular surgery and reticuloendothelium system, in Rob C, Smith R (eds): Clinical Surgery, vol 14. Philadelphia, PA, Lippincott, 1967, pp 140-141 Schwartz HC, Kendrick RW, Pogrel BS: False aneurysm of the matiary artery: An unusual complication of closed facial trauma. Arch Otolaryngol109:616, 1983 Cohen MA: False (traumatic) aneurysm of the facial artery caused by a foreign body. Int J Oral Maxillofac Surg 15:336, 1986 Kennedy JW, Kent JN: False aneurysm and a partial facial paralysis secondary to mandibuiar fracture: Report of case. J Oral Surg 28:854, 1970 Taylor DV: Traumatic aneurysm and facial palsy as complication of a mandibular fracture. Br J Oral Surg 4:202, 1967 Calem WS: Traumatic (false) aneurysm of the terminal portion of the external carotid artery. Am J Surg 106:522, 1963 Wagner M: Pseudoaneurysm. A complication of percutaneous angiography and angiocardiography. JAMA 186:427, 1963 Derdeyn CP, Moran CJ, Cross DT, et al: Intraoperative digital subtraction angiography: a review of 112 consecutive examinations. AJNR 16:307, 1995 Heiserman JE, Dean BL, Hodak JA, et al: Neurologic complications of cerebral angiography. AJNR 15:1401, 1994


Journal of Oral and Maxillofacial Surgery | 2008

Orthognathic Surgery in the Young Cleft Patient: Preliminary Study on Subsequent Facial Growth

Larry M. Wolford; Daniel Serra Cassano; David A. Cottrell; Mohamed El Deeb; Spiro C Karras; João Roberto Gonçalves

PURPOSE This study evaluated the long-term effects of orthognathic surgery on subsequent growth of the maxillomandibular complex in the young cleft patient. PATIENTS AND METHODS We evaluated 12 young cleft patients (9 male and 3 female patients), with a mean age of 12 years 6 months (range, 9 years 8 months to 15 years 4 months), who underwent Le Fort I osteotomies, with maxillary advancement, expansion, and/or downgrafting, by use of autogenous bone or hydroxyapatite grafts, when indicated, for maxillary stabilization. Five patients had concomitant osteotomies of the mandibular ramus. All patients had presurgical and postsurgical orthodontic treatment to control the occlusion. Radiographs taken at initial evaluation (T1) and presurgery (T2) were compared to establish the facial growth vector before surgery, whereas radiographs taken immediately postsurgery (T3) and at longest follow-up (T4) were used to determine postsurgical growth. Each patients lateral cephalograms were traced, and 16 landmarks were identified and used to compute 11 measurements describing presurgical and postsurgical growth. RESULTS Before surgery, all patients had relatively normal growth. After surgery, cephalograms showed statistically significant growth changes from T3 to T4, with the maxillary depth decreasing by -3.3 degrees +/- 1.8 degrees , Sella-nasion-point A by -3.3 degrees +/- 1.8 degrees, and point A-nasion-point B by -3.6 degrees +/- 2.8 degrees. The angulation of the maxillary incisors increased by 9.2 degrees +/- 11.7 degrees. Of 12 patients, 11 showed disproportionate postsurgical jaw growth. Maxillary growth occurred predominantly in a vertical vector with no anteroposterior growth, even though most patients had shown anteroposterior growth before surgery. The distance increased in the linear measurement from nasion to gnathion by 10.3 +/- 7.9 mm. Four of 5 patients operated on during the mixed dentition phase had teeth that erupted through the cleft area. A variable impairment of postoperative growth was seen with the 2 types of grafting material used. No significant difference was noted in the effect on growth in patients with unilateral clefts versus those with bilateral clefts. The presence of a pharyngeal flap was noted to adversely affect growth, whereas simultaneous mandibular surgery did not. After surgery, 11 of 12 patients tended toward a Class III end-on occlusal relation. CONCLUSIONS Orthognathic surgery may be performed on growing cleft patients when mandated by psychological and/or functional concerns. The surgeon must be cognizant of the adverse postsurgical growth outcomes when performing orthognathic surgery on growing cleft patients with the possibility for further surgery requirements. Performing maxillary osteotomies on cleft patients would be more predictable after completion of facial growth.


American Journal of Orthodontics and Dentofacial Orthopedics | 1997

Condylar change after upward and forward rotation of the maxillomandibular complex

David A. Cottrell; Roberto M. Suguimoto; Larry M. Wolford; Rohit C.L. Sachdeva; Ingrid Y. Guo

Tomographic radiographs of 44 healthy temporomandibular joints in 22 patients undergoing large upward and forward rotations of the maxillomandibular complex were evaluated. Immediate postsurgery results showed decreases in all areas of the joint space. Long-term results showed a decrease in the superior and posterior regions, 14.8% and 19.7%, with no significant change anteriorly. Simple and multiple regression analysis did not show any relationship with age, gender, or degree of change in the decrease of the occlusal plane angle, to the temporomandibular joint changes described. Some condylar remodeling was noted.


International Journal of Oral and Maxillofacial Surgery | 2013

Efficacy of anti-inflammatory drugs in third molar surgery: a randomized clinical trial.

Pushkar Mehra; U. Reebye; Mohammed Nadershah; David A. Cottrell

This was a double-blind randomized clinical trial to assess the effect of different pharmacological regimens on the level of prostaglandin E2 (PGE2) in urine and saliva, and to correlate the findings to the clinical course after removal of impacted lower third molars. Eighty patients were randomly divided into four groups: group 1 received placebo; group 2 received preoperative ibuprofen, which was continued for a week; group 3 received intraoperative dexamethasone; and group 4 received preoperative ibuprofen, which was continued for a week, in addition to intraoperative dexamethasone. Saliva and urine samples were taken at scheduled intervals. Patients receiving ibuprofen fared significantly better in most parameters. A single dose of dexamethasone alone had a potent but transient beneficial effect when compared to the results with ibuprofen, which showed significant improvement in both subjective and objective parameters. Use of a single dose of intravenous steroids perioperatively helped reduce untoward sequelae, although to a lesser degree and for a shorter duration than continuous ibuprofen. Combining ibuprofen with perioperative dexamethasone added some benefit in some of the measured parameters, but without a statistically significant advantage over using ibuprofen only.


Journal of Oral and Maxillofacial Surgery | 2000

Painless Mass in the Parotid Region

John E. Mullins; Orrett E. Ogle; David A. Cottrell

A 12-year-old Hispanic girl presented to the oral and maxillofacial surgery clinic at Woodhull Medical Center with a chief complaint of a nontender right facial swelling. The history was obtained from both the father and the patient through a translator. This revealed that the patient first became aware of the swelling about 4 months previously and that it had continued to increase in size. On further questioning about the origin of the swelling, the father gave a history of a mild to moderate upper respiratory infection at about the same time that the swelling first appeared. The medical history indicated that the child had not received the usual childhood vaccines. She was never hospitalized, was not on any medications, and did receive routine pediatric care. Overall, her general health had been good. The social history disclosed that the patient moved from Mexico approximately 1 year ago and was living with her father, aunt, uncle, and 2 siblings, ages 5 and 18 years, in a 3-bedroom apartment in Brooklyn, New York. The mother was still living in Mexico with her remaining 2 children, ages 14 and 15 years. The patient had achieved the fourth grade while in Mexico, but had never attended school in the United States. She denied having a boyfriend; nor had she had any close personal or sexual contact with anyone in the United States. There was no history of tobacco, alcohol, or illicit drug usage. The review of systems revealed that she had had 3 or 4 upper respiratory infections since coming to the United States; there was no history of chronic cough or hemoptosis. She had not yet reached menarche. Examination showed a 5.0 3.5-cm swelling located over the center of the right parotid gland (Fig 1). The skin was erythematous, and there was a sinus tract located at the inferior aspect from which a serosanguinous exudate was draining. On palpation, the swelling was noted to be fixed, fluctuant, and was not warm to touch. The submandibular and cervical lymph nodes were palpable and nontender. Cranial nerves II to XII were grossly intact. The rest of the head and neck examination was within normal limits. The intraoral examination showed a mixed dentition in good repair. The oral mucosa, tongue, floor of mouth, and throat appeared normal. Stenson’s duct was patent and produced clear saliva on milking. Results of the rest of the physical examination were normal. The patient did not have an elevated temperature. The panoramic radiograph of the jaws showed a pattern that was normal for a 12-year-old child.


Journal of Oral and Maxillofacial Surgery | 1995

Infraorbital nerve sharing to restore sensibility to the lower lip: Case report

Larry M. Wolford; David A. Cottrell; John P. LaBanc

43. Klinar KL, McManis JC: Soft-tissue ameloblastoma: Report of a case. Oral Surg 28:266, 1969 44. Wesley RK, Borninski ER, Mintz S: Peripheral ameloblastoma: Report of case and review of literature. J Oral Surg 35:670, 1977 45. Moskow BS, Baden E: The peripheral ameloblastoma of the gingiva: Case report and literature review. J Periodontol 53:736, 1982 46. Zhu EX: Dental lamina as presumptive source of odontogenic cyst. J Stomatol Soc Jpn 57:549, 1990 47. Gardner DG: Peripheral ameloblastoma: A study of 21 cases, including 5 reported as basal cell carcinoma of the gingiva. Cancer 39:1625, 1977 48. Stanley HR, Krogh HW: Peripheral ameloblastoma: Report of a case. Oral Surg Oral Med Oral Pathol 12:760, 1959 49. Russell A: Ameloblastoma of mucosal origin. N Z Dent J 62:116, 1966 50. Wertheimer FW, Stroud DE: Peripheral ameloblastoma in a papilloma with recurrence: Report of case. J Oral Surg 30:47, 1972 51. Wallen NG: Extraosseous ameloblastoma. Oral Surg 34:95, 1972 52. Simpson HE: Basal cell carcinoma and peripheral ameloblastoma. Oral Surg 38:233, 1974 53. Richardson JF, Greer RO: Ameloblastoma of mucosal origin: A pathologic reevaluation. Arch Otolaryngol 100:174, 1974 54. Pansino FA, Meara JW: Peripheral ameloblastoma. J Mich Dent Assoc 57:129, 1975 55. Greer RO, Hammond WS: Extraosseous ameloblastoma: Light microscopic and ultrastructural observations. J Oral Surg 36:553, 1978 56. Gould AR, Farman AG, DeJean EK, et al: Peripheral ameloblastoma: An ultrastructural analysis. J Oral Pathol 11:90, 1982 57. Guralnick W, Chuong R, Goodman M: Peripheral ameloblastoma of the gingiva. J Oral Maxillofac Surg 41:536, 1983 58. Schaberg S J, Antimarino RF, Pierce GL, et al: Peripheral ameloblastoma: Report of a case. Int J Oral Surg 12:344, 1983 59. Anneroth G, Johansson B: Peripheral ameloblastoma. Int J Oral Surg 14:295, 1985 60. Braunstein E: Case report of an extraosseous adamantinoblastoma. Oral Surg 2:276, 1949 61. Ficarra G, Hansen LS: Peripheral ameloblastoma: A case report. J Craniomaxillofac Surg 15:110, 1987 62. Horowitz I, Hirshberg A, Dayan D: Peripheral ameloblastoma: A clinical dilemma in gingiva lesions. J Clin Periodontal 14:366, 1987

Collaboration


Dive into the David A. Cottrell's collaboration.

Top Co-Authors

Avatar
Top Co-Authors

Avatar
Top Co-Authors

Avatar
Top Co-Authors

Avatar
Top Co-Authors

Avatar
Top Co-Authors

Avatar
Top Co-Authors

Avatar
Top Co-Authors

Avatar
Researchain Logo
Decentralizing Knowledge