David M. Reed

Predictors of Use of Services Among Dementia Caregivers

Caregivers of persons with dementia do not use community resources until late in the disease process, despite the fact that judicious use of community resources can delay nursing home admission. Data from the National Caregiver Training Project, based on Hall and Buckwalter’s (1987) progressively lowered stress threshold (PLST) model, were used to examine variables related to use of community resources. Spouse and adult child caregivers were divided into two groups based on amount of community resources used per week. Within this geographically diverse sample of caregivers, 64% did not use professional services, 79% did not use respite services, and 65% did not use other services. Being a spouse decreased the odds that the caregiver would use community resources. Resource use was also related to the care recipient’s problems with activities of daily living and the increase in frequency of memory and behavioral problems.

Bones of the Maya: Studies of Ancient Skeletons

Includes an indexed bibliography of the first 150 years of Maya osteology. This volume pulls together a spectrum of bioarchaeologists that reveal remarkable data on Maya genetic relationship, demography, and diseases.

Who's the boss? Family/staff partnership in care of persons with dementia

This article provides an overview of family involvement in care intervention and its implementation with African American and Caucasian family members of persons with dementia in nursing home settings.

A Locus for Posterior Polymorphous Corneal Dystrophy (PPCD3) Maps to Chromosome 10

Posterior polymorphous corneal dystrophy (PPCD) is an autosomal dominant disorder characterized by corneal endothelial abnormalities, which can lead to blindness due to loss of corneal transparency and sometimes glaucoma. We mapped a new locus responsible for PPCD in a family in which we excluded the previously reported PPCD locus on 20q11, and the region containing COL8A2 on chromosome 1. Results of a 317‐marker genome scan provided significant evidence of linkage of PPCD to markers on chromosome 10, with single‐point LOD scores of 2.63, 1.63, and 3.19 for markers D10S208 (at

Decisional involvement: staff nurse and nurse manager perceptions.

\hat \theta = 0.03

MAP Kinase Pathway is Involved in IGF-1-Stimulated Proliferation of Human Retinal Pigment Epithelial Cells (hRPE)

), D10S1780 (at

Intravitreal Daptomycin: A Safety and Efficacy Study

\hat \theta = 0.00

Evaluation of the reliability and validity of nursing outcomes classification patient outcomes and measures

), and D10S578 (at

ERK-Mediated Activation of Fas Apoptotic Inhibitory Molecule 2 (Faim2) Prevents Apoptosis of 661W Cells in a Model of Detachment-Induced Photoreceptor Cell Death

\hat \theta = 0.06

Nursing Diagnoses, Interventions, and Patient Outcomes for Hospitalized Older Adults with Pneumonia

). A maximum multi‐point LOD score of 4.35 was found at marker D10S1780. Affected family members shared a haplotype in an 8.55 cM critical interval that was bounded by markers D10S213 and D10S578. Our finding of another PPCD locus, PPCD3, on chromosome 10 indicates that PPCD is genetically heterogeneous. Guttae, a common corneal finding sometimes observed along with PPCD, were found among both affected and unaffected members of the probands sib ship, but were absent in the younger generations of the family. Evaluation of phenotypic differences between family members sharing the same affected haplotype raises questions about whether differences in disease severity, including differences in response to surgical interventions, could be due to genetic background or other factors independent of the PPCD3 locus.