Fang Ding-ye

Hydrogen sulfide poisoning of methanol synthesis Cu-based catalyst. I: Intrinsic deactivation kinetics

Abstract The hydrogen sulfide poisoning of the methanol synthesis C207Cu-based catalyst was investigated at atmospheric pressure, temperature 250 ∼265°C,hydrogen sulfide content 100–280 ppm, based on the methanol composition reaction. The higher the hydrogen sulfide content, the faster the rate of deactivation; the higher the temperature, the faster the rate of deactivation. The intrinsic deactivation rate equations were derived from the experimental data. In addition, the poisoned catalyst was measured by X-ray diffraction.

Comparison of FeMn, FeMnNa and FeMnK Catalysts for the Preparation of Light Olefins from Syngas

The influence of sodium and potassium promoters on the structure and reaction behavior of an FeMn catalyst toward light olefin synthesis from syngas was investigated by N2 adsorption, X-ray photoelectron spectroscopy (XPS), X-ray diffraction (XRD), H2 temperature-programmed reduction (H2-TPR), CO/CO2 temperature-programmed desorption (CO/CO2-TPD), Mossbauer spectroscopy (MES) and CO+H2 reaction. We found that an increase in manganese improves the dispersion of the active Fe component and light olefin selectivity; however, excessive enrichment with the Mn promoter on the catalyst surface suppresses CO conversion. Potassium and sodium inhibit the reduction of the catalyst in H2 and improve the adsorption of CO2 and CO because of the enhanced surface basicity of the catalysts. After reduction with syngas (nH2/nCO=20) and reaction with syngas (nH2/nCO=3.5), the analysis of the bulk structure was compared with those of the FeMn, FeMnNa, and FeMnK catalysts. The results show that FeCx is found in relatively high levels in the FeMnK 1932 LI Jiang-Bing et al.: Comparison of FeMn, FeMnNa and FeMnK Catalysts for the Preparation of Light Olefins No.10

HYDROGEN SULFIDE POISONING OF METHANOL SYNTHESIS Cu-BASED CATALYST∗ (III) EFFECTIVENESS FACTOR OF POISONED CATALYST

Abstract The single component model and the multicomponent model of the effectiveness factor ζd, of the poisoned catalyst have been proposed and solved by the orthogonal collocation method. The center equilibrium dead zone was founded in catalyst pellets. The calculated values ζd, were respectively 0·1229–0·2907,0·1298–0·2445 and 0·0690–0·2155 for No. 1, No. 2 and No. 3 catalyst. The average of the absolute values of the relative deviations between the experimental values and the calculated values by model for ζd was less than 11 per cent. The models can be used to calculate the effectiveness factor of the poisoned catalyst.

Global kinetic models of methanol synthesis in three-phase agitated slurry reactor

Abstract The global kinetic models of methanol synthesis from carbon monoxide, carbon dioxide and hydrogen are investigated in the three-phase well mixed reactor with mechanical agitator under such conditions: pressure 5 MPa, temperature 210∼250°C, methanol synthesis catalyst C301, medicinal grade liquid paraffin as inert medium, rotation speed of agitator 950 rpm. By the improved Gauss-Newton method, the global kinetic models are developed.

HYDROGEN SULFIDE POISONING OF METHANOL SYNTHESIS Cu-BASED CATALYST∗(II’) POISONING OF COMMERCIAL CATALYST

Abstract In the absence of H2S and with the addition of inlet H2S content 719 ppm for 4 hours, 8 hours and 12 hours respectively, the global reaction rates of methanol decomposition were determined over φ 5×5 mm cylindrical pellet of C207 Cu -based catalyst at atmospheric pressure in an internal recycle gradientless reactor. The pore size distribution, the profile of sulfur in pellet, and the tortuosity factor of the poisoned catalyst were also measured. The hydrogen sulfide poisoning of commercial catalyst was considered as the shell-progressive poisoning. The experimental values of the non-poisoned dimensionless radius xc by Auger Electron Spectroscopy were 0·938, 0·916 and 0·896 respectively with the addition of H2S for 4 hours, 8 hours and 12 hours, which were in agreement with the values calculated by the intrinsic deactivation kinetics.