Ian V. Lau

A VISCOUS TOLERANCE CRITERION FOR SOFT TISSUE INJURY ASSESSMENT

Experiments in our laboratory have documented that high-speed impact can cause severe injury to internal organs before either of the currently accepted chest injury criteria, which are based on spinal acceleration or chest compression, approach their tolerance limit. Those studies demonstrate an interdependence between the velocity of deformation and compression of the body on injury risk. A tolerable level of chest compression at a low velocity can prove to be fatal at higher velocities of deformation. The observation of a rate-sensitive tolerable compression led to the introduction of the Viscous criterion, VCmax, which accounts for the importance of both parameters. VCmax is the maximum of the product of velocity of deformation (V) and compression (C), and is derivable from the chest deflection response. This paper presents the empirical evidence and theoretical basis supporting the Viscous criterion, and shows it to be an indicator of the energy dissipated by soft tissue deformation. The Viscous criterion accurately predicts the risk of vital organ and soft tissue injury when other criteria fail.

BIOMECHANICS OF THE HUMAN CHEST, ABDOMEN, AND PELVIS IN LATERAL IMPACT

Fourteen unembalmed cadavers were subjected to 44 blunt lateral impacts at velocities of approximately 4.5, 6.7, or 9.4 m/s with a 15 cm flat circular interface on a 23.4 kg pendulum accelerated to impact speed by a pneumatic impactor. Chest and abdominal injuries consisted primarily of rib fractures, with a few cases of lung or liver laceration in the highest severity impacts. There were two cases of pubic ramus fracture in the pelvic impacts. Logist analysis of the biomechanical responses and injury indicated that the maximum Viscous response had a slightly better correlation with injury than maximum compression for chest and abdominal impacts. A tolerance level of VC = 1.47 m/s for the chest and VC = 1.98 m/s for the abdomen were determined for a 25% probability of critical injury. Maximum compression was similarly set at C = 38% for the chest and at C = 44% for the abdomen. The experiments indicate that chest and abdominal injury may occur by a viscous mechanism during the rapid phase of body compression, and that the Viscous and compression responses are effective, complementary measures of injury risk in side impact. Although serious pelvic injury was infrequent, lateral public ramus fracture correlated with compression of the pelvis, not impact force or pelvic acceleration. Pelvic tolerance was set at 27% compression.

MECHANISM OF INJURY FROM AIR BAG DEPLOYMENT LOADS

Loadings induced by deploying currently representative air bags were studied with driver surrogates (anesthetized swine) leaning against the system during inflation. Torso injury mechanisms were studied in a physiologic model, supported against a static steering wheel-mounted air bag system. Severe and extensive chest and abdominal injuries to the swine were observed in the tests. Loading caused by air bag deployment can occur in either of two phases. The first phase represents the initial punch out of the bag from the module; the second phase represents the membrane force of the inflating bag. Statistical analysis indicated that punch out induced injury because of the high rate of loading to the surrogate body region in direct contact with the air bag module. Membrane forces induced injury by high compression over a larger area. Punch-out loading might be reduced by allowing the bag to escape from other parts of the container not in contact with the driver during deployment. Loading by the inflating bag might be reduced by using a compliant steering system to support the module. The amount and rate of generated gas had only marginal effect on the cumulative injury. Even an inflator with inadequate gas output to protect a properly seated occupant had sufficient energy to induce severe injuries in a surrogate in contact with the inflating module. Analysis of the field relevance of the results must consider not only the injury potential given that a driver is in direct contact with the air bag module at the time of deployment, but also the expected field frequency of such an event. Analysis of the field relevance of the results must also consider the correlation of the laboratory test environment with real-world exposure.

Biomechanics of Liver Injury by Steering Wheel Loading

Abdominal injury induced by steering wheel contact at a velocity of 32 km/hr was investigated using anesthetized swine as the surrogate on a Hyge sled. The lower rim of the wheel was positioned 5 cm below the xyphoid. By varying wheel stiffness, wheel orientation, and column angle, resultant abdominal injury ranged from fatal or critical to minor or none. Wheel stiffness was found to be the primary determinant of abdominal injury severity. The mechanism of abdominal injury was identified to be the rim impacting the abdomen and exceeding a combined velocity and compression sensitive tolerance limit. Abdominal injury occurred within the initial 15 ms of wheel contact before whole body movement of the surrogate of column compression, which were initiated by hub contact with the thorax. The severity of abdominal injury correlated with the peak viscous response which can be represented by the product of the instantaneous velocity of abdominal deformation and abdominal compression. It did not correlate with spinal acceleration.

BIOMECHANICS OF INJURY IN LATERAL IMPACTS

Fourteen anesthetized swine were subjected to blunt lateral impact at velocities of 4.3, 6.7, or 8.2 m/s with a 15 cm flat pendulum weighing 23.4 kg accelerated to impact speed by a power-assisted pneumatic impactor. Injuries consisted of laceration of the liver and spleen resulting in severe hemoperitoneum and death by ventricular fibrillation and respiratory arrest in the highest severity impacts. Logist analysis of the biomechanical responses and serious or fatal injury indicated that the maximum Viscous response (VC) had the best correlation with injury risk. A tolerance level of VC = 0.89 m/s was determined for a 25% probability of serious injury. In contrast, maximum chest compression did not correlate with injury. The experiments indicate that internal organ and soft tissue injury may occur by a Viscous mechanism during the rapid phase of compression of the body. The Viscous response is an effective measure of injury risk in side impacts.