J A Janovick

Brain gonadotropin releasing hormone receptors: localization and regulation.

Stable transfection of GH3 cells with rat gonadotropin-releasing hormone receptor complementary deoxyribonucleic acid results in expression of a receptor coupled to cyclic adenosine 3',5'-monophosphate-dependent prolactin release via a G-protein.

A cholera toxin-sensitive guanyl nucleotide binding protein mediates the movement of pituitary luteinizing hormone into a releasable pool: loss of this event is associated with the onset of homologous desensitization to gonadotropin-releasing hormone.

SIIp: A Unique Secretogranin/Chromogranin of the Pituitary Released in Response to Gonadotropin- Releasing Hormone*

Gonadotropin-releasing hormone-stimulated intracellular Ca2+ fluctuations and luteinizing hormone release can be uncoupled from inositol phosphate production

Differential orientation of a GnRH agonist and antagonist in the pituitary GnRH receptor.

Caldesmon: A Bifunctional (Calmodulin and Actin) Binding Protein Which Regulates Stimulated Gonadotropin Release*

Gonadotropin-Releasing Hormone Modulation of Protein Kinase-C Activity in Per if used Anterior Pituitary Cell Cultures

GH3 cells transfected with gonadotropin-releasing hormone (GnRH) receptor complementary deoxyribonucleic acid release secretogranin-II through a constitutive pathway after GnRH analog-regulated synthesis: evidence that secretory proteins do not contain a sequence that obligates processing through a secretory granule or by regulated secretion

Selection of models for the study of GnRH stimulated gonadotropin release prejudices the assignment of roles for mediators and modulators of hormone action