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Experimental Biology and Medicine | 1950

Relation of Cortisone Pretreatment to Mobilization of Lipids to Liver by Pituitary Extracts.

Louis Levin; Rita K. Farber

Summary and conclusions 1, Many, but not all, pituitary preparations produce an increase in liver fat content when administered to intact mice. This response is completely abolished by adrenalectomy. 2. Neither cortisone nor any other adrenocortical steroid tested to date is able, of itself, to cause a substantial increase in liver fat level of intact or of adrenalectomized mice. In the latter, however, the usual post-adren-alectomy loss of liver fat is prevented. 3. When pre-treated with adrenocortical hormone (cortisone), the ability of adrenalectomized mice to respond to pituitary hormone by mobilization of fat to the liver is equal to or greater than that of intact mice. Pituitary substances (growth hormone; weakly potent ACTH) inactive in either intact or adrenalectomized animals, became quite effective in mobilizing fat to the liver if the animals are pre-treated with adrenocortical hormone. 4. It is concluded that to mobilize fat from the depots to the liver the animal requires at least two factors, (1) adequate supplies of adrenocortical hormone (from either endogenous or exogenous sources) plus (2) a supply, either from its own anterior hypophysis or from exogenous sources, of the “triggering” pituitary factor. 5. The “triggering” action of the unidentified pituitary factor is not mediated via the adrenal cortex since it operates in the absence of the adrenal gland provided adequate amounts of exogenous cortisone are administered. 6. The possible identity of the “triggering” pituitary factor is discussed. The authors are indebted to Miss Ilse Bauer for her kind assistance in performing the many adrenalectomies and necropsies.


Endocrinology | 1937

THE QUANTITATIVE ASSAY OF “FOLLICLE STIMULATING” SUBSTANCES1

Louis Levin; H. H. Tyndale


Annals of the New York Academy of Sciences | 1951

SECTION OF BIOLOGY: SYMPOSIUM RECENT DEVELOPMENTS IN ENDOCRINOLOGY: THE ENDOCRINE MECHANISM CONTROLLING THE MOBILIZATION OF DEPOT FAT*†

Louis Levin; Rita K. Farber


Endocrinology | 1945

THE EFFECTS OF SEVERAL VARIETIES OF STRESS ON THE CHOLESTEROL CONTENT OF THE ADRENAL GLANDS AND OF THE SERUM OF RATS1

Louis Levin


Endocrinology | 1941

QUANTITATIVE PRECIPITATION OF THE URINARY GONADOTROPIN OF NORMAL MEN AND WOMEN1

Louis Levin


Endocrinology | 1948

THE EFFECT OF INDUCED SECRETORY ACTIVITY ON THE CHOLESTEROL CONTENT OF THE IMMATURE RAT OVARY1

Louis Levin; Joseph W. Jailer


American Journal of Obstetrics and Gynecology | 1949

On the Validity of the Hyperemia Method for Determining Ovulation Time in Women

Louis Levin; C.L. Buxton; Earl T. Engle


Endocrinology | 1938

HORMONE STUDIES WITH THE ULTRA-CENTRIFUGE II. THE CONCENTRATION OF ANTERIOR LOBE AND PITUITARY-LIKE HORMONES WITH THE ULTRA-CENTRIFUGE1

Aura E. Severinghaus; Louis Levin; J.A. Chiles


Endocrinology | 1938

IS THE BLOOD CALCIUM LEVEL OF MAMMALS INFLUENCED BY ESTROGENIC SUBSTANCES?1

Louis Levin; Philip E. Smith


Endocrinology | 1941

THE PHENOMENON OF ANTAGONISM BY PITUITARY EXTRACT IN THE ABSENCE OF THE THYROID GLAND1

George K. Smelser; Louis Levin

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