Mehmet Eren Yuksel
Aksaray University
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Featured researches published by Mehmet Eren Yuksel.
Indian Journal of Dermatology | 2018
Funda Tamer; Mehmet Eren Yuksel
Sample size is a not an issue in this study. The study was designed to be done during the summer months. We recruited all AA patients during our study period (May to October). We did not calculate sample size for our study apriori. However, the study results reveal that our study had a good precision and a large effect size. The 95% confidence interval for the difference between Vitamin D levels among the AA patients versus the controls was −10.9 ng/ml–−6.9 ng/ml in our study.[2]
Dermatology practical & conceptual | 2018
Funda Tamer; Mehmet Eren Yuksel; Evren Sarifakioglu; Yavuz Karabag
Background Seborrheic dermatitis is an inflammatory skin disease that affects 1–3% of the general population. The Malassezia species has been implicated as the main causative agent; however, the bacterial flora of the skin may also play role in the etiopathogenesis. Therefore, we investigated the most common bacterial agent of the skin flora of patients with seborrheic dermatitis. Materials and Methods Fifty-one patients with seborrheic dermatitis and 50 healthy individuals are included in this study. Sterile cotton swabs were rubbed on the scalp of the participants for bacterial culture. Colonial morphology was identified with gram stain and catalase test. Results Staphylococcus aureus was isolated from 25 (49%) patients with seborrheic dermatitis and 10 (20%) healthy individuals within the control group. Coagulase-negative staphylococci were isolated from 24 (47.1%) patients with seborrheic dermatitis and 17 (34%) healthy individuals within the control group. Diphtheroids were present in 2 (3.9%) patients and 1 (2%) subject within the control group. Gram-negative bacilli were present only in 1 (2%) patient. Hemolytic streptococci and bacilli were identified in 1 (2%) subject from each group. Colonization of coagulase-negative staphylococci, diphtheroids, gram-negative bacilli, hemolytic streptococci, and bacillus did not differ between patients and healthy controls. However, S. aureus colonization was significantly more common in patients with seborrheic dermatitis than in healthy controls. Conclusion Within this study we revealed that S. aureus colonization was significantly higher among the patients. Therefore, we propose that, in addition to the Malassezia species, S. aureus may play a role in the etiopathogenesis of seborrheic dermatitis.
Our Dermatology Online | 2016
Mehmet Eren Yuksel; Funda Tamer
A 61-year-old Caucasian male presented with a breast lump for futher clinical evaluation. The patient admitted that the slowly growing mass had been present for a year. The lesion was asymptomatic, however the patient was concerned about the risk of malignancy. The past medical history and family history were both unremarkable. The physical examination of the patient revealed a skin coloured, well-defined, firm, subcutaneous nodule with a punctum and telangiectasias on the surface (Fig. 1). The lesion was located on the trunk, laterally to the right areola measuring 3x2.5 cm. There was no axillary lymphadenopathy. The lesion was surgically removed without any complications (Fig. 2). The histopathological examination of the specimen revealed an epidermal cyst.
Indian Journal of Dermatology, Venereology and Leprology | 2016
Funda Tamer; Mehmet Eren Yuksel
1. Miller SA, Dykes DD, Polesky HF. A simple salting out procedure for extracting DNA from human nucleated cells. Nucleic Acids Res 1988;16:1215. 2. Lamartine J, Munhoz Essenfelder G, Kibar Z, Lanneluc I, Callouet E, Laoudj D, et al. Mutations in GJB6 cause hidrotic ectodermal dysplasia. Nat Genet 2000;26:142‐4. 3. Smith FJ, Morley SM, McLean WH. A novel connexin 30 mutation in Clouston syndrome. J Invest Dermatol 2002;118:530‐2. 4. Baris HN, Zlotogorski A, Peretz‐Amit G, Doviner V, Shohat M, Reznik‐Wolf H, et al. A novel GJB6 missense mutation in hidrotic ectodermal dysplasia 2 (Clouston syndrome) broadens its genotypic basis. Br J Dermatol 2008;159:1373‐6. 5. Liu YT, Guo K, Li J, Liu Y, Zeng WH, Geng SM. Novel mutations in GJB6 and GJB2 in Clouston syndrome. Clin Exp Dermatol 2015;40:770‐3. 6. Kellermayer R, Keller M, Ratajczak P, Richardson E, Harangi F, Mérei E, et al. Bigenic connexin mutations in a patient with hidrotic ectodermal dysplasia. Eur J Dermatol 2005;15:75‐9. 7. Radhakrishna U, Blouin JL, Mehenni H, Mehta TY, Sheth FJ, Sheth JJ, et al. The gene for autosomal dominant hidrotic ectodermal dysplasia (Clouston syndrome) in a large Indian family maps to the 13q11‐q12.1 pericentromeric region. Am J Med Genet 1997;71:80‐6.
Our Dermatology Online | 2017
Funda Tamer; Mehmet Eren Yuksel
European Medical, Health and Pharmaceutical Journal | 2015
Mehmet Eren Yuksel; Funda Tamer
Our Dermatology Online | 2018
Funda Tamer; Mehmet Eren Yuksel
Our Dermatology Online | 2018
Funda Tamer; Mehmet Eren Yuksel
Dermatology Online Journal | 2018
Mehmet Eren Yuksel; Funda Tamer; Nurten Bozlak
Romanian Journal of Clinical and Experimental Dermatology | 2017
Funda Tamer; Mehmet Eren Yuksel; Haldun Umudum