Mitch Lomax

Inspiratory muscle warm-up and inspiratory muscle training: separate and combined effects on intermittent running to exhaustion

Abstract In the present study, we examined the independent and combined effects of an inspiratory muscle warm-up and inspiratory muscle training on intermittent running to exhaustion. Twelve males were recruited to undertake four experimental trials. Two trials (Trials 1 and 2) preceded either a 4-week training period of 1 × 30 breaths twice daily at 50% (experimental group) or 15% (control group) maximal inspiratory mouth pressure (PImax). A further two trials (Trials 3 and 4) were performed after the 4 weeks. Trials 2 and 4 were preceded by a warm-up: 2 × 30 breaths at 40% PImax. Pre-training PImax and distance covered increased (P < 0.05) similarly between groups after the warm-up (∼11% and ∼5–7% PImax and distance covered, respectively). After training, PImax increased by 20 ± 6.1% (P < 0.01; d = 3.6) and 26.7 ± 6.3% (P < 0.01; d = 3.1) when training and warm-up were combined in the experimental group. Distance covered increased after training in the experimental group by 12 ± 4.9% (P < 0.01; d = 3.6) and 14.9 ± 4.5% (P < 0.01; d = 2.3) when training and warm-up interventions were combined. In conclusion, inspiratory muscle training and inspiratory muscle warm-up can both increase running distance independently, but the greatest increase is observed when they are combined.

Inspiratory muscle fatigue significantly affects breathing frequency, stroke rate, and stroke length during 200-m front-crawl swimming.

Lomax, M and Castle, S. Inspiratory muscle fatigue significantly affects breathing frequency, stroke rate, and stroke length during 200-m front-crawl swimming. J Strength Cond Res 25(10): 2691–2695, 2011—The aim of the current study was to assess the impact of inspiratory muscle fatigue (IMF) on total breaths taken (ftot), breaths per minute (fb), stroke count (SC), stroke rate (SR), and stroke length (SL) during constant velocity front-crawl swimming. Eight collegiate swimmers undertook a 200-m front-crawl swim on 2 separate occasions. On 1 occasion, IMF was induced immediately before the swim (IMF trial), and on the other occasion, the swim was undertaken in the absence of IMF (control trial). Trials were administered using a randomized crossover design and at a swimming velocity equivalent to 85% of race pace: Pilot testing identified this as being the fastest pace, which did not induce IMF. Maximal inspiratory mouth pressure, which was measured at the mouth and from residual volume, fell by 17% (p < 0.05) in response to IMF but was unchanged in response to the swim itself (p < 0.05). When compared to the control trial, ftot, fb, SC, and SR increased (p < 0.05) and SL decreased (p < 0.05) in response to IMF. These data suggest that the increase in ftotand fb in the presence of IMF occurred, in part, in an attempt to alleviate dyspnea. As a result, SL decreased and SR and SC increased, although variability in the SR and SC response did occur. However, as a number of identical muscles are recruited during deep inspirations and the front-crawl arm stroke, the possibility that arm coordination was changed, in part, to compensate for a reduced force-generating capacity per arm stroke should not be overlooked.

The effect of three recovery protocols on blood lactate clearance after race-paced swimming.

Abstract Lomax, M. The effect of three recovery protocols on blood lactate clearance after race-paced swimming. J Strength Cond Res 26(10): 2771–2776, 2012—The purpose of the present study was to assess the impact of 3 recovery protocols on blood lactate clearance after maximal intensity swimming. Thirty-three regional standard swimmers were tested throughout the course a year and were required to complete a race-paced 200-m swim in their main stroke or individual medley. After the race-paced swim, swimmers were assigned a self-paced continuous steady rate swim of 20 minutes (self-prescribed); a 20-minute coach-administered modified warm-up consisting of various swimming modes, intensities, and rest intervals (coach prescribed); or a 20-minute land-based recovery consisting of light-intensity walking, skipping, and stretching (land based). Blood lactate concentration was measured from the fingertip before and after the race-paced swim and after the recovery activity. The concentration of blood lactate was higher (p < 0.01) after race-paced swimming (range of 10.5–11.0 mmol·L−1) compared with baseline (range 1.3–1.4 mmol·L−1). However, there were no differences (p > 0.05) between the groups (recovery protocols) at these time points. Conversely, differences were observed between groups after the recovery activities (p < 0.01). Specifically, blood lactate concentration was higher after the land-based activity (3.7 ± 1.8 mmol·L−1) than either the self-prescribed (2.0 ± 1.2 mmol·L−1) or coach-prescribed (1.8 ± 0.9 mmol·L−1) swimming protocols. The results of the present study suggest that it does not matter whether a self-paced continuous steady rate swimming velocity or a swimming recovery consisting of various strokes, intensities, and rest intervals is adopted as a recovery activity. As both swimming recoveries removed more blood lactate than the land-based recovery, swimmers should therefore be advised to undertake a swimming-based recovery rather than a land-based recovery.

Inspiratory Muscle Fatigue After Race-Paced Swimming Is Not Restricted to the Front Crawl Stroke

Abstract Lomax, M, Iggleden, C, Tourell, A, Castle, S, and Honey, J. Inspiratory muscle fatigue following race-paced swimming is not restricted to the front crawl stroke. J Strength Cond Res 26(10): 2729–2733, 2012—The occurrence of inspiratory muscle fatigue (IMF) has been documented after front crawl (FC) swimming of various distances. Whether IMF occurs after other competitive swimming strokes is not known. The aim of the present study was to assess the impact of all 4 competitive swimming strokes on the occurrence of IMF after race-paced swimming and to determine whether the magnitude of IMF was related to the breathing pattern adopted and hence breathing frequency (fb). Eleven, nationally ranked, youth swimmers completed four 200-m swims (one in each competitive stroke) on separate occasions. The order of the swims, which consisted of FC, backstroke (BK), breaststroke (BR), and butterfly (FLY), was randomized. Maximal inspiratory mouth pressure (MIP) was assessed before (after a swimming and inspiratory muscle warm-up) and after each swim with fb calculated post swim from recorded data. Inspiratory muscle fatigue was evident after each 200-m swim (p < 0.05) but did not differ between the 4 strokes (range 18–21%). No relationship (p > 0.05) was observed between fb and the change in MIP (FC: r = −0.456; BK: r = 0.218; BR: r = 0.218; and FLY: r = 0.312). These results demonstrate that IMF occurs in response to 200-m race-paced swimming in all strokes and that the magnitude of IMF is similar between strokes when breathing is ad libitum occurring no less than 1 breath (inhalation) every third stroke.

An electromyographic evaluation of dual role breathing and upper body muscles in response to front crawl swimming

The upper body trunk musculature is key in supporting breathing, propulsion, and stabilization during front crawl swimming. The aim of this study was to determine if the latissimus dorsi, pectoralis major, and serratus anterior contributed to the development of inspiratory muscle fatigue observed following front crawl swimming. Fourteen trained swimmers completed a 200‐m front crawl swim at 90% of race pace. Maximal inspiratory and expiratory mouth pressures (PImax and PEmax) were assessed before (baseline) and after each swim, and electromyography was recorded from the three muscles. Post‐swim PImax fell by 11% (P < 0.001, d = 0.57) and the median frequency (MDF: a measure of fatigue) of the latissimus dorsi, pectoralis major, and serratus anterior fell to 90% (P = 0.001, d = 1.57), 87% (P = 0.001, r = −0.60) and 89% (P = 0.018, d = 1.04) of baseline, respectively. The fall in serratus anterior MDF was correlated with breathing frequency (r = 0.675, P = 0.008) and stroke rate (r = 0.639, P = 0.014). The results suggest that the occurrence of inspiratory muscle fatigue was partly caused by fatigue of these muscles, and that breathing frequency and stroke rate particularly affect the serratus anterior.

Airway dysfunction in elite swimmers: prevalence, impact and challenges

The prevalence of airway dysfunction in elite swimmers is among the highest in elite athletes. The traditional view that swimmers naturally gravitate toward swimming because of preexisting respiratory disorders has been challenged. There is now sufficient evidence that the higher prevalence of bronchial tone disorders in elite swimmers is not the result of a natural selection bias. Rather, the combined effects of repeated chlorine by-product exposure and chronic endurance training can lead to airway dysfunction and atopy. This review will detail the underpinning causes of airway dysfunction observed in elite swimmers. It will also show that airway dysfunction does not prevent success in elite level swimming. Neither does it inhibit lung growth and might be partially reversible when elite swimmers retire from competition.

Inspiratory muscle fatigue affects latissimus dorsi but not pectoralis major activity during arms only front crawl sprinting.

Abstract Lomax, M, Tasker, L, and Bostanci, O. Inspiratory muscle fatigue affects latissimus dorsi but not pectoralis major activity during arms only front crawl sprinting. J Strength Cond Res 28(8): 2262–2269, 2014—The purpose of this study was to determine whether inspiratory muscle fatigue (IMF) affects the muscle activity of the latissimus dorsi and pectoralis major during maximal arms only front crawl swimming. Eight collegiate swimmers were recruited to perform 2 maximal 20-second arms only front crawl sprints in a swimming flume. Both sprints were performed on the same day, and IMF was induced 30 minutes after the first (control) sprint. Maximal inspiratory and expiratory mouth pressures (PImax and PEmax, respectively) were measured before and after each sprint. The median frequency (MDF) of the electromyographic signal burst was recorded from the latissimus dorsi and pectoralis major during each 20-second sprint along with stroke rate and breathing frequency. Median frequency was assessed in absolute units (Hz) and then referenced to the start of the control sprint for normalization. After IMF inducement, stroke rate increased from 56 ± 4 to 59 ± 5 cycles per minute, and latissimus dorsi MDF fell from 67 ± 11 Hz at the start of the sprint to 61 ± 9 Hz at the end. No change was observed in the MDF of the latissimus dorsi during the control sprint. Conversely, the MDF of the pectoralis major shifted to lower frequencies during both sprints but was unaffected by IMF. As the latter induced fatigue in the latissimus dorsi, which was not otherwise apparent during maximal arms only control sprinting, the presence of IMF affects the activity of the latissimus dorsi during front crawl sprinting.

Cold water swimming and upper respiratory tract infections

It is often suggested that habitual cold water swimming (HCS) may improve resistance to infection [1], yet research into effects of HCS on the immune system has produced inconclusive results. This may be due to the wide range of protocols, from brief ice-cold dips [2] to long cold water swims [3]. Many studies measured blood and saliva markers rather than actual illness, and the clinical significance of these markers is not well established [4]. Incidence of upper respiratory tract infection (URTI), i.e. the common cold, is a useful indication of in vivo immune system function [5,6]. This study compared URTI susceptibility in those practising HCS with that in their non-swimming co-habiting partners. To control for any effect of swimming, those who swim in indoor heated pools and their partners were also investigated. The null hypothesis (H0) was that there would be no difference between swimming groups.

Scientific rationale for changing lower water temperature limits for triathlon racing to 12°C with wetsuits and 16°C without wetsuits

Objectives To provide a scientific rationale for lower water temperature and wetsuit rules for elite and subelite triathletes. Methods 11 lean, competitive triathletes completed a 20 min flume swim, technical transition including bike control and psychomotor testing and a cycle across five different wetsuit and water temperature conditions: with wetsuit: 10°C, 12°C and 14°C; without wetsuit (skins): 14°C and 16°C. Deep body (rectal) temperature (Tre), psychomotor performance and the ability to complete a technical bike course after the swim were measured, as well as swimming and cycling performance. Results In skins conditions, only 4 out of 11 athletes could complete the condition in 14°C water, with two becoming hypothermic (Tre<35°C) after a 20 min swim. All 11 athletes completed the condition in 16°C. Tre fell further following 14°C (mean 1.12°C) than 16°C (mean 0.59°C) skins swim (p=0.01). In wetsuit conditions, cold shock prevented most athletes (4 out of 7) from completing the swim in 10°C. In 12°C and 14°C almost all athletes completed the condition (17 out of 18). There was no difference in temperature or performance variables between conditions following wetsuit swims at 12°C and 14°C. Conclusion The minimum recommended water temperature for racing is 12°C in wetsuits and 16°C without wetsuits. International Triathlon Union rules for racing were changed accordingly (January 2017).

Impact of weekly swimming training distance on the ergogenicity of inspiratory muscle training in well trained youth swimmers.

Lomax, M, Kapus, J, Brown, PI, and Faghy, M. Impact of weekly swimming training distance on the ergogenicity of inspiratory muscle training in well-trained youth swimmers. J Strength Cond Res 33(8): 2185-2193, 2019-The aim of this study was to examine the impact of weekly swimming training distance on the ergogenicity of inspiratory muscle training (IMT). Thirty-three youth swimmers were recruited and separated into a LOW and HIGH group based on weekly training distance (≤31 km·wk and >41 km·wk, respectively). The LOW and HIGH groups were further subdivided into control and IMT groups for a 6-week IMT intervention giving a total of 4 groups: LOWcon, LOWIMT, HIGHcon, and HIGHIMT. Before and after the intervention period, swimmers completed maximal effort 100- and 200-m front crawl swims, with maximal inspiratory and expiratory mouth pressures (PImax and PEmax, respectively) assessed before and after each swim. Inspiratory muscle training increased PImax (but not PEmax) by 36% in LOWIMT and HIGHIMT groups (p ≤ 0.05), but 100- and 200-m swims were faster only in the LOWIMT group (3 and 7% respectively, p ≤ 0.05). Performance benefits only occurred in those training up to 31 km·wk and indicate that the ergogenicity of IMT is affected by weekly training distance. Consequently, training distances are important considerations, among others, when deciding whether or not to supplement swimming training with IMT.