Robert S. Ormond
Henry Ford Hospital
Network
Latest external collaboration on country level. Dive into details by clicking on the dots.
Publication
Featured researches published by Robert S. Ormond.
Radiology | 1971
Robert S. Ormond; Melvyn Rubenfire; Daniel T. Anbe; Ellet H. Drake
Abstract The epicardial fat defines the limit of the myocardium. It can be regularly visualized using image intensification and recorded on spot-films obtained by a special technique. The relationship between the electrode of the transvenous pacemaker and the epicardial fat indicates the relationship between the electrode and the myocardium. If the separation is less than 3 mm, penetration has occurred. Of 33 patients examined, 11 demonstrated some degree of penetration.
Radiology | 1966
Robert S. Ormond; Henry H. Gale; Ellet H. Drake; Thomas Gahagan
In the past twenty-four months we have engaged in a program to encourage the performance of pulmonary angiography in suspected cases of life-threatening pulmonary embolism. Pulmonary embolectomy is performed with or without the aid of the heart lung bypass (1), following clipping (2) or ligation of the inferior vena cava in patients with occlusion of more than 60 per cent of the pulmonary arterial bed. Anticoagulant therapy, with or without inferior vena cava interruption, is administered to patients with lesser obstruction. If pulmonary embolism is excluded, appropriate studies are performed to establish the proper diagnosis, and indicated therapy is instituted. Treatment which may have been directed toward embolism is discontinued. The following cases illustrate the advantages of pulmonary angiography. Case I: A 50-year-old woman in critical condition with the diagnosis of pulmonary embolism was transferred from an outside hospital. Four weeks previously resection of a small bowel hemangioma had been pe...
Radiology | 1964
Robert S. Ormond; Ellet H. Drake; Henry H. Gale
Systemic embolization of left atrial thrombi is a dread complication of mitral stenosis which is particularly likely to occur during and immediately following surgery on the mitral valve. The threat of embolization is confined to patients with atrial fibrillation. Taber and Lam (4) reported from this institution (The Henry Ford Hospital, Detroit) operative or postoperative embolization in only 2 patients of a group of 226 with normal sinus rhythm, compared to 29 operative or postoperative embolizations in 200 patients with atrial fibrillation. In 41 of the 200 patients preoperative embolization was present, and in 11 of these, emboli were noted in the operative or immediate postoperative period. The presence of an atrial thrombus is a contraindication for closed mitral commissurotomy, and unless all patients with fibrillation are to be operated upon by open heart technics the preoperative determination of the presence or absence of thrombi assumes importance. The angiographic demonstration of left atrial ...
American Journal of Surgery | 1966
Thomas Gahagan; Henry H. Gale; Robert S. Ormond
Abstract Pulmonary angiograms are carried out in all patients suspected of having pulmonary embolism. The decision for treatment is based on (1) the condition of the patient, (2) the pulmonary arterial pressure, and (3) the amount of pulmonary arterial obstruction as shown by angiograms. If the patient has normal vital signs without dyspnea and cyanosis, no elevation of pulmonary arterial pressure, and only a small portion of the pulmonary vascular bed occluded, caval interruption only is indicated. If the vital signs are altered, the pulmonary arterial pressure is elevated, and there is more than 60 per cent occlusion of the pulmonary arterial tree, immediate embolectomy with cardiopulmonary bypass is indicated.
Radiology | 1967
Robert S. Ormond; Ellet H. Drake; Frank J. Hildner
The primary pathologic changes in pulmonary hypertension occur in muscular arteries measuring less than 1,000 microns in diameter. Heath and Edwards (3) divided the progressive vascular changes into six grades, with all but the first occurring in all forms of hypertensive disease, including primary pulmonary hypertension. In severe forms of hypertensive vascular disease there is progressive occlusion of vessels measuring less than 500 microns, while arteries larger than 600 microns show intimal fibrosis. Excessive dilatation occurs in some of the smaller muscular arteries, with the formation of microaneurysms in the most marked form of the disease. Short of lung biopsy, pulmonary angiography is the most direct method of studying the anatomy of the pulmonary vasculature. The smallest vessels that can be visualized on angiograms, however, are only slightly less than 500 microns in diameter, and thus the majority of the specific changes occurring in hypertension are invisible. Secondary changes in the larger...
JAMA | 1967
Frank J. Hildner; Robert S. Ormond
Chest | 1973
Melvyn Rubenfire; Daniel T. Anbe; Ellet H. Drake; Robert S. Ormond
American Journal of Cardiology | 1966
Frank J. Hildner; Ellet H. Drake; Henry H. Gale; Robert S. Ormond
Chest | 1966
Conrad R. Lam; Rodman E. Taber; Henry H. Gale; Robert S. Ormond
American Journal of Cardiology | 1967
Frank J. Hildner; William R. Pierson; Bernard Weinstock; Ellet H. Drake; Robert S. Ormond