Vincent T. Martin
University of Cincinnati
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Publication
Featured researches published by Vincent T. Martin.
Headache | 2006
Vincent T. Martin; Michael M. Behbehani
Migraine headache is strongly influenced by reproductive events that occur throughout the lifespan of women. Each of these reproductive events has a different “hormonal milieu,” which might modulate the clinical course of migraine headache. Estrogen and progesterone can be preventative or provocative for migraine headache under different circumstances depending on their absolute serum levels, constancy of exposure, and types of estrogen/progesterone derivatives. Attacks of migraine with and without aura respond differently to changes in ovarian hormones. Clearly a greater knowledge of ovarian hormones and their effect on migraine is essential to a greater understanding of the mechanisms and pathogenesis of migraine headache.
Headache | 2004
David W. Dodick; Richard B. Lipton; Vincent T. Martin; Vasilios Papademetriou; Wayne D. Rosamond; Antoinette Maassen VanDenBrink; Hassan Loutfi; K. Michael Welch; Peter J. Goadsby; Steven R. Hahn; Susan Hutchinson; David B. Matchar; Stephen D. Silberstein; Timothy R. Smith; R. Allan Purdy; Jane Saiers
Background.—Health care providers frequently cite concerns about cardiovascular safety of the triptans as a barrier to their use. In 2002, the American Headache Society convened the Triptan Cardiovascular Safety Expert Panel to evaluate the evidence on triptan‐associated cardiovascular risk and to formulate consensus recommendations for making informed decisions for their use in patients with migraine.
Headache | 2010
Gretchen E. Tietjen; Jan Lewis Brandes; B. Lee Peterlin; Arnolda Eloff; Rima M. Dafer; Michael R. Stein; Ellen Drexler; Vincent T. Martin; Susan Hutchinson; Sheena K. Aurora; Ana Recober; Nabeel A. Herial; Christine Utley; Leah White; Sadik A. Khuder
(Headache 2010;50:20‐31)
Headache | 2009
Gretchen E. Tietjen; Jan Lewis Brandes; B. Lee Peterlin; Arnolda Eloff; Rima M. Dafer; Michael R. Stein; Ellen Drexler; Vincent T. Martin; Susan Hutchinson; Sheena K. Aurora; Ana Recober; Nabeel A. Herial; Christine Utley; Leah White; Sadik A. Khuder
Background.— Cutaneous allodynia (CA) in migraine is a clinical manifestation of central nervous system sensitization. Several chronic pain syndromes and mood disorders are comorbid with migraine. In this study we examine the relationship of migraine‐associated CA with these comorbid conditions. We also evaluate the association of CA with factors such as demographic profiles, migraine characteristics, and smoking status that may have an influence on the relationships of CA to pain and mood.
Medical Clinics of North America | 2001
Vincent T. Martin; Michael M. Behbehani
The typical migraine patient is exposed to a myriad of migraine triggers on a daily basis. These triggers potentially can act at various sites within the cerebral vasculature and the central nervous system to promote the development of migraine headache. The challenge to the physician is in the identification and avoidance of migraine trigger factors within patients suffering from migraine headache. Only through a rational approach to migraine trigger factors can physicians develop an appropriate treatment strategy for migraine patients.
Headache | 2010
Gretchen E. Tietjen; Jan Lewis Brandes; B. Lee Peterlin; Arnolda Eloff; Rima M. Dafer; Michael R. Stein; Ellen Drexler; Vincent T. Martin; Susan Hutchinson; Sheena K. Aurora; Ana Recober; Nabeel A. Herial; Christine Utley; Leah White; Sadik A. Khuder
(Headache 2010;50:42‐51)
Headache | 2010
Gretchen E. Tietjen; Jan Lewis Brandes; B. Lee Peterlin; Arnolda Eloff; Rima M. Dafer; Michael R. Stein; Ellen Drexler; Vincent T. Martin; Susan Hutchinson; Sheena K. Aurora; Ana Recober; Nabeel A. Herial; Christine Utley; Leah White; Sadik A. Khuder
(Headache 2010;50:32‐41)
Headache | 2007
Gretchen E. Tietjen; B. Lee Peterlin; Jan Lewis Brandes; Faizan Hafeez; Susan Hutchinson; Vincent T. Martin; Rima M. Dafer; Sheena K. Aurora; Michael R. Stein; Nabeel A. Herial; Christine Utley; Leah White; Sadik A. Khuder
Objective.—To discern the effects of depression and anxiety on the migraine–obesity relationship.
Headache | 2008
Vincent T. Martin; Richard B. Lipton
Migraine is frequently associated with menstruation in female migraineurs, and consequently it is commonly referred to as menstrually associated migraine. The trigger thought to be partially responsible for menstrually associated migraine is a significant drop in circulating estrogen that is noted during 2‐3 days prior to onset of menses. It is estimated that approximately 50% of women have an increased risk of experiencing migraine during the premenstrual phase of decreasing estrogen levels. Understanding the biological basis of migraine associated with menses will facilitate an accurate diagnosis and help patients recognize time susceptible to migraine exacerbations. This paper will review the biological bases for the hormonal changes that occur during the menstrual cycle and review the prevalence and burden of menstrual migraine among female headache sufferers.
Headache | 2005
Kathleen A. Foley; Roger K. Cady; Vincent T. Martin; James U. Adelman; Merle L. Diamond; Christopher F. Bell; Jeffrey M. Dayno; X. Henry Hu
Introduction.—Although research suggests that early treatment of migraine headache when the pain is mild results in better outcomes for patients, many patients delay taking their acute‐migraine medication until their headaches are moderate or severe. Understanding when and why patients use their migraine medications is an important first step to improve migraine management.