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Dive into the research topics where Vivian Maria Arakelian is active.

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Featured researches published by Vivian Maria Arakelian.


Clinical Physiology and Functional Imaging | 2011

The measurement of lactate threshold in resistance exercise: a comparison of methods.

Nuno Manuel Frade de Sousa; Rodrigo Ferro Magosso; Guilherme Borges Pereira; Richard Diego Leite; Vivian Maria Arakelian; Arlindo N Montagnolli; Sérgio Eduardo de Andrade Perez; Vilmar Baldissera

Resistance incremental tests (IT) make it possible to determine critical metabolic and cardiovascular changes, such as the lactate threshold (LT). Different methods are frequently used to improve the exactness of LT identification. The objective of the study was to identify LT by four different methods (visual inspection, log–log, algorithmic adjustment and QLac) during resistance exercise and to evaluate which methods present more precision. Twelve men performed a maximal IT on the leg press at relative intensities of 10%, 20%, 25%, 30%, 35%, 40%, 50%, 60%, 70%, 80% and 90% of 1RM with 1‐min stages. During the 2‐min interval between stages, capillary blood was collected for blood lactate analysis. LT was detected using each of the four methods. The intensity of LT by visual inspection method was 26·9 (5·2)% of 1RM, adjustment algorithmic method was 27·8 (3·6)% of 1RM, log–log method was 23·3 (3·5)% of 1RM and QLac method was 31·6 (9·8)% of 1RM, with significant difference only between log–log and QLac methods. Bland and Altman analysis shows better concordance for visual inspection versus adjustment algorithmic methods. The visual inspection, algorithmic and log–log methods detected the LT at the same intensity. The mathematical models, specially the algorithmic method, provide more precision.


American Journal of Physical Medicine & Rehabilitation | 2016

Effects of Light-Emitting Diode Therapy on Muscle Hypertrophy, Gene Expression, Performance, Damage, and Delayed-Onset Muscle Soreness: Case-control Study with a Pair of Identical Twins.

Cleber Ferraresi; Danilo Rodrigues Bertucci; Josiane Lilian dos Santos Schiavinato; Rodrigo Reiff; Amélia G. Araújo; Rodrigo A. Panepucci; Euclides Matheucci; Anderson Ferreira da Cunha; Vivian Maria Arakelian; Michael R. Hamblin; Nivaldo Antonio Parizotto; Vanderlei Salvador Bagnato

ObjectiveThe aim of this study was to verify how a pair of monozygotic twins would respond to light-emitting diode therapy (LEDT) or placebo combined with a strength-training program during 12 weeks. DesignThis case-control study enrolled a pair of male monozygotic twins, allocated randomly to LEDT or placebo therapies. Light-emitting diode therapy or placebo was applied from a flexible light-emitting diode array (&lgr; = 850 nm, total energy = 75 J, t = 15 seconds) to both quadriceps femoris muscles of each twin immediately after each strength training session (3 times/wk for 12 weeks) consisting of leg press and leg extension exercises with load of 80% and 50% of the 1-repetition maximum test, respectively. Muscle biopsies, magnetic resonance imaging, maximal load, and fatigue resistance tests were conducted before and after the training program to assess gene expression, muscle hypertrophy and performance, respectively. Creatine kinase levels in blood and visual analog scale assessed muscle damage and delayed-onset muscle soreness, respectively, during the training program. ResultsCompared with placebo, LEDT increased the maximal load in exercise and reduced fatigue, creatine kinase, and visual analog scale. Gene expression analyses showed decreases in markers of inflammation (interleukin 1&bgr;) and muscle atrophy (myostatin) with LEDT. Protein synthesis (mammalian target of rapamycin) and oxidative stress defense (SOD2 [mitochondrial superoxide dismutase]) were up-regulated with LEDT, together with increases in thigh muscle hypertrophy. ConclusionsLight-emitting diode therapy can be useful to reduce muscle damage, pain, and atrophy, as well as to increase muscle mass, recovery, and athletic performance in rehabilitation programs and sports medicine.


American Journal of Physical Medicine & Rehabilitation | 2017

Hemodynamic, Autonomic, Ventilatory, and Metabolic Alterations After Resistance Training in Patients With Coronary Artery Disease: A Randomized Controlled Trial

Flávia Cristina Rossi Caruso; José Carlos Bonjorno; Ross Arena; Shane A. Phillips; Ramona Cabiddu; Renata Gonçalves Mendes; Vivian Maria Arakelian; Daniela Bassi; Audrey Borghi-Silva

Objective The aim of this work was to evaluate the hemodynamic, autonomic, and metabolic responses during resistance and dynamic exercise before and after an 8-week resistance training program using a low-intensity (30% of 1 repetitium maximum), high-repetition (3 sets of 20 repetitions) model, added to an aerobic training program, in a coronary artery disease cohort. Design Twenty male subjects with coronary artery disease (61.1 ± 4.7 years) were randomly assigned to a combined training group (resistance + aerobic) or aerobic training group (AG). Heart rate, stroke volume, cardiac output, minute ventilation, blood lactate, and parasympathetic modulation indices of heart rate (square root of the mean squared differences of successive RR intervals [RMSSD] and dispersion of points perpendicular to the line of identity that provides information about the instantaneous beat-to-beat variability [SD1]) were obtained before and after an 8-week RT program while performing exercise on a cycle ergometer and a 45-degree leg press. Results Resistance training resulted in an increase in maximal and submaximal load tolerance (P < 0.01), a decreased hemodynamic response (P < 0.01), and a reduction in blood lactate in the combined training group compared to the aerobic training group during the 45-degree leg press. During exercise on a cycle ergometer, there was a decreased hemodynamic response and increased minute ventilation (P < 0.01). The 8-week RT program resulted in greater parasympathetic tone (RMSSD and SD1) and an increase in the SDNN index during exercise on a cycle ergometer and 45-degree leg press (P < 0.05). Conclusions An 8-week resistance training program associated with aerobic training may attenuate hemodynamic stress, and modify metabolic and autonomic responses during resistance exercise. The training program also appeared to elicit beneficial cardiovascular and autonomic effects during exercise.


Revista Brasileira De Medicina Do Esporte | 2015

Poor glycemic control impacts linear and non-linear dynamics of heart rate in DM type 2

Daniela Bassi; Vivian Maria Arakelian; Renata Gonçalves Mendes; Flávia Cristina Rossi Caruso; José Carlos Bonjorno Júnior; Katiany Thays Lopes Zangrando; Cláudio Ricardo de Oliveira; Jacob M. Haus; Ross Arena; Audrey Borghi-Silva

INTRODUCAO: E de conhecimento geral que o diabetes mellitus tipo 2 (DM2) produz neuropatia autonomica cardiovascular (NAC), que pode afetar a modulacao autonomica cardiaca. Entretanto, nao e claro se a falta de controle glicemico em diabeticos tipo 2 sem NAC, poderia impactar negativamente na modulacao autonomica cardiaca. Objetivo: Avaliar a relacao entre controle glicemico e modulacao autonomica cardiaca em individuos com DM2 sem neuropatia autonomica cardiovascular. Estudo descritivo, prospectivo e transversal. METODOS: Quarenta e nove pacientes com DM2 (51±7 anos) foram divididos em dois grupos de acordo com a hemoglobina glicosilada (HbA1c): G1: ≤ 7% e G2: >7,0%. A frequencia cardiaca de repouso (FC) e intervalo RR (iRR) foram obtidos e calculados por metodos lineares (media iRR; media FC; rMSSD; STD RR; LF; HF; LF/HF, TINN e RR Tri) e nao lineares (SD1; SD2; DFα1; DFα2, Entropia de Shannon; ApEn; SampEn e CD) de variabilidade de frequencia cardiaca. Insulina, HOMA-IR, glicemia de jejum e HbA1c foram obtidas por analises sanguineas. RESULTADOS: G2 (HbA1c ≤ 7%) mostrou valores menores para media de iRR; STD RR; RR Tri, TINN, SD2, CD e maiores para media de FR quando comparado com G1 (HbA1c > 7%). Adicionalmente, HbA1c correlacionou-se negativamente com media iRR (r=0,28, p=0,044); STD RR (r=0,33, p=0,017); RR Tri (r=-0,35, p=0,013), SD2 (r=-0,39, p=0,004) e positivamente com media FC (r=0,28, p=0,045). Finalmente, a glicemia de jejum correlacionou-se negativamente com STD RR (r=-0,36, p=0,010); RR Tri (r=-0,36, p=0,010); TINN (r=-0,33, p=0,019) e SD2 (r=-0,42, p=0,002). CONCLUSAO: Conclui que o controle glicemico deficiente relaciona-se com indices de modulacao autonomica cardiaca em individuos com DM2, ainda que nao apresentem neuropatia autonomica cardiovascular.INTRODUCCION: Es de conocimiento general que la diabetes mellitus tipo 2 (DM2) produce neuropatia autonomica cardiovascular (NAC), que puede afectar la modulacion autonomica cardiaca. Entretanto, no es claro si la falta de control glucemico en diabeticos tipo 2 sin NAC, podria impactar negativamente en la modulacion autonomica cardiaca. Objetivo: Evaluar la relacion entre control glucemico y modulacion autonomica cardiaca en individuos con DM2 sin neuropatia autonomica cardiovascular. Estudio descriptivo, prospectivo y transversal. METODOS: Cuarenta y nueve pacientes con DM2 (51±7 anos) fueron divididos en dos grupos de acuerdo con la hemoglobina glucosilada (HbA1c): G1: ≤ 7% y G2: >7,0%. La frecuencia cardiaca de reposo (FC) e intervalo RR (iRR) fueron obtenidos y calculados por metodos lineales (promedio iRR; promedio FC; rMSSD; STD RR; LF; HF; LF/HF, TINN y RR Tri) y no lineales (SD1; SD2; DFα1; DFα2, Entropia de Shannon; ApEn; SampEn y CD) de variabilidad de frecuencia cardiaca. Fueron obtenidas insulina, HOMA-IR, glucemia en ayunas y HbA1c a traves de analisis sanguineos. RESULTADOS: G2 (HbA1c ≤ 7%) mostro valores menores para el promedio de iRR; STD RR; RR Tri, TINN, SD2, CD y mayores para el promedio de FR al ser comparado con G1 (HbA1c > 7%). Adicionalmente, HbA1c se correlaciono negativamente con el promedio iRR (r=0,28, p=0,044); STD RR (r=0,33, p=0,017); RR Tri (r=-0,35, p=0,013), SD2 (r=-0,39, p=0,004) y positivamente con el promedio FC (r=0,28, p=0,045). Finalmente, la glucemia en ayunas se correlaciono negativamente con STD RR (r=-0,36, p=0,010); RR Tri (r=-0,36, p=0,010); TINN (r=-0,33, p=0,019) e SD2 (r=-0,42, p=0,002). CONCLUSION: Concluimos que el control glucemico deficiente se relaciona con indices de modulacion autonomica cardiaca en individuos con DM2, aunque no presenten neuropatia autonomica cardiovascular.


Journal of Sports Medicine and Physical Fitness | 2016

Critical load: A novel approach to determining a sustainable intensity during resistance exercise

Vivian Maria Arakelian; Renata Gonçalves Mendes; Renata Trimer; Flávia Cristina Rossi Caruso; Nuno Mf de Sousa; Vanessa C. Borges; Camila do Valle Gomes Gatto; Vilmar Baldissera; Ross Arena; Audrey Borghi-Silva

BACKGROUND A hyperbolic function as well as a linear relationship between power output and time to exhaustion (Tlim) has been consistently observed during dynamic non-resistive exercises. However, little is known about its concept to resistance exercises (RE), which could be defined as critical load (CL). This study aimed to verify the existence of CL during dynamic RE and to verify the number of workbouts necessary to determine the optimal modeling to achieve it. METHODS Fifteen healthy men (23±2.5 yrs) completed 1 repetition maximum test (1RM) on a leg press and 3 (60%, 75% and 90% of 1RM) or 4 (+ 30% of 1RM) workbouts protocols to obtain the CL by hyperbolic and linear regression models between Tlim and load performed. Blood lactate and leg fatigue were also measured. RESULTS CL was obtained during RE and 3 workbouts protocol estimate it at 53% while 4 tests at 38% of 1 RM. However, based on coefficients of determination, 3 protocols provided a better fit than the 4-parameter model, respectively (R2>0.95 vs. >0.77). Moreover, all intensities increased blood lactate and leg fatigue, however, when corrected by Tlim, were significantly lower at CL. CONCLUSIONS It was possible to determinate CL during dynamic lower limbs RE and that 3 exhaustive workbouts can be used to better estimate the CL, constituting a new concept of determining this threshold during dynamic RE and reducing the physically demanding nature of the protocol. These findings may have important applications for functional performance evaluation and prescription of RE programs.


Arquivos Brasileiros De Cardiologia | 2018

Efeitos da Coexistência de Diabetes Tipo 2 e Hipertensão sobre a Variabilidade da Frequência Cardíaca e Capacidade Cardiorrespiratória

Daniela Bassi; Ramona Cabiddu; Renata Gonçalves Mendes; Natália Barbosa Tossini; Vivian Maria Arakelian; Flávia Cristina Rossi Caruso; José Carlos Bonjorno Junior; Ross Arena; Audrey Borghi-Silva

Background Type 2 diabetes Mellitus (T2DM) is associated with cardiac autonomic dysfunction, which is an independent predictor of mortality in chronic diseases. However, whether the coexistence of systemic arterial hypertension (HTN) with DMT2 alters cardiac autonomic modulation remains unknown. Objective To evaluate the influence of HTN on cardiac autonomic modulation and cardiorespiratory fitness in subjects with DMT2. Methods 60 patients of both genders were evaluated and allocated to two groups: DMT2 patients (n = 32; 51 ± 7.5 years old) and DMT2 + HTN patients (n = 28; 51 ± 6.9 years old). RR intervals were obtained during rest in supine position. Linear and nonlinear indices of heart rate variability (HRV) were computed using Kubios HRV software. Pulmonary gas exchange was measured breath-by-breath, using a portable telemetric system during maximal incremental exercise testing on a cycle ergometer. Statistical analysis included Shapiro-Wilk test followed by Student’s t Test, Pearson correlation and linear regression. Results We found that patients in the DMT2+HTN group showed lower values of mean RR intervals (801.1 vs 871.5 ms), Shannon entropy (3 vs 3.2) and fractal dimension SD 1 (9.5 vs 14.5), when contrasted with patients in the DMT2 group. Negative correlations were found between some HRV nonlinear indices and exercise capacity indices. Conclusion HTN negatively affects the cardiac autonomic function in diabetic patients, who are already prone to develop autonomic dysfunction. Strategies are need to improve cardiac autonomic functionality in this population.


Revista Brasileira De Medicina Do Esporte | 2015

Pobre controle glicêmico impacta a dinâmica linear e não linear da frequência cardíaca no DM2

Daniela Bassi; Vivian Maria Arakelian; Renata Gonçalves Mendes; Flávia Cristina Rossi Caruso; José Carlos Bonjorno Júnior; Katiany Thays Lopes Zangrando; Cláudio Ricardo de Oliveira; Jacob M. Haus; Ross Arena; Audrey Borghi-Silva

INTRODUCAO: E de conhecimento geral que o diabetes mellitus tipo 2 (DM2) produz neuropatia autonomica cardiovascular (NAC), que pode afetar a modulacao autonomica cardiaca. Entretanto, nao e claro se a falta de controle glicemico em diabeticos tipo 2 sem NAC, poderia impactar negativamente na modulacao autonomica cardiaca. Objetivo: Avaliar a relacao entre controle glicemico e modulacao autonomica cardiaca em individuos com DM2 sem neuropatia autonomica cardiovascular. Estudo descritivo, prospectivo e transversal. METODOS: Quarenta e nove pacientes com DM2 (51±7 anos) foram divididos em dois grupos de acordo com a hemoglobina glicosilada (HbA1c): G1: ≤ 7% e G2: >7,0%. A frequencia cardiaca de repouso (FC) e intervalo RR (iRR) foram obtidos e calculados por metodos lineares (media iRR; media FC; rMSSD; STD RR; LF; HF; LF/HF, TINN e RR Tri) e nao lineares (SD1; SD2; DFα1; DFα2, Entropia de Shannon; ApEn; SampEn e CD) de variabilidade de frequencia cardiaca. Insulina, HOMA-IR, glicemia de jejum e HbA1c foram obtidas por analises sanguineas. RESULTADOS: G2 (HbA1c ≤ 7%) mostrou valores menores para media de iRR; STD RR; RR Tri, TINN, SD2, CD e maiores para media de FR quando comparado com G1 (HbA1c > 7%). Adicionalmente, HbA1c correlacionou-se negativamente com media iRR (r=0,28, p=0,044); STD RR (r=0,33, p=0,017); RR Tri (r=-0,35, p=0,013), SD2 (r=-0,39, p=0,004) e positivamente com media FC (r=0,28, p=0,045). Finalmente, a glicemia de jejum correlacionou-se negativamente com STD RR (r=-0,36, p=0,010); RR Tri (r=-0,36, p=0,010); TINN (r=-0,33, p=0,019) e SD2 (r=-0,42, p=0,002). CONCLUSAO: Conclui que o controle glicemico deficiente relaciona-se com indices de modulacao autonomica cardiaca em individuos com DM2, ainda que nao apresentem neuropatia autonomica cardiovascular.INTRODUCCION: Es de conocimiento general que la diabetes mellitus tipo 2 (DM2) produce neuropatia autonomica cardiovascular (NAC), que puede afectar la modulacion autonomica cardiaca. Entretanto, no es claro si la falta de control glucemico en diabeticos tipo 2 sin NAC, podria impactar negativamente en la modulacion autonomica cardiaca. Objetivo: Evaluar la relacion entre control glucemico y modulacion autonomica cardiaca en individuos con DM2 sin neuropatia autonomica cardiovascular. Estudio descriptivo, prospectivo y transversal. METODOS: Cuarenta y nueve pacientes con DM2 (51±7 anos) fueron divididos en dos grupos de acuerdo con la hemoglobina glucosilada (HbA1c): G1: ≤ 7% y G2: >7,0%. La frecuencia cardiaca de reposo (FC) e intervalo RR (iRR) fueron obtenidos y calculados por metodos lineales (promedio iRR; promedio FC; rMSSD; STD RR; LF; HF; LF/HF, TINN y RR Tri) y no lineales (SD1; SD2; DFα1; DFα2, Entropia de Shannon; ApEn; SampEn y CD) de variabilidad de frecuencia cardiaca. Fueron obtenidas insulina, HOMA-IR, glucemia en ayunas y HbA1c a traves de analisis sanguineos. RESULTADOS: G2 (HbA1c ≤ 7%) mostro valores menores para el promedio de iRR; STD RR; RR Tri, TINN, SD2, CD y mayores para el promedio de FR al ser comparado con G1 (HbA1c > 7%). Adicionalmente, HbA1c se correlaciono negativamente con el promedio iRR (r=0,28, p=0,044); STD RR (r=0,33, p=0,017); RR Tri (r=-0,35, p=0,013), SD2 (r=-0,39, p=0,004) y positivamente con el promedio FC (r=0,28, p=0,045). Finalmente, la glucemia en ayunas se correlaciono negativamente con STD RR (r=-0,36, p=0,010); RR Tri (r=-0,36, p=0,010); TINN (r=-0,33, p=0,019) e SD2 (r=-0,42, p=0,002). CONCLUSION: Concluimos que el control glucemico deficiente se relaciona con indices de modulacion autonomica cardiaca en individuos con DM2, aunque no presenten neuropatia autonomica cardiovascular.


Revista Brasileira De Medicina Do Esporte | 2015

Control glucémico deficiente impacta la dinámica lineal y no lineal de la frecuencia cardíaca en el DM2

Daniela Bassi; Vivian Maria Arakelian; Renata Gonçalves Mendes; Flávia Cristina Rossi Caruso; José Carlos Bonjorno Júnior; Katiany Thays Lopes Zangrando; Cláudio Ricardo de Oliveira; Jacob M. Haus; Ross Arena; Audrey Borghi-Silva

INTRODUCAO: E de conhecimento geral que o diabetes mellitus tipo 2 (DM2) produz neuropatia autonomica cardiovascular (NAC), que pode afetar a modulacao autonomica cardiaca. Entretanto, nao e claro se a falta de controle glicemico em diabeticos tipo 2 sem NAC, poderia impactar negativamente na modulacao autonomica cardiaca. Objetivo: Avaliar a relacao entre controle glicemico e modulacao autonomica cardiaca em individuos com DM2 sem neuropatia autonomica cardiovascular. Estudo descritivo, prospectivo e transversal. METODOS: Quarenta e nove pacientes com DM2 (51±7 anos) foram divididos em dois grupos de acordo com a hemoglobina glicosilada (HbA1c): G1: ≤ 7% e G2: >7,0%. A frequencia cardiaca de repouso (FC) e intervalo RR (iRR) foram obtidos e calculados por metodos lineares (media iRR; media FC; rMSSD; STD RR; LF; HF; LF/HF, TINN e RR Tri) e nao lineares (SD1; SD2; DFα1; DFα2, Entropia de Shannon; ApEn; SampEn e CD) de variabilidade de frequencia cardiaca. Insulina, HOMA-IR, glicemia de jejum e HbA1c foram obtidas por analises sanguineas. RESULTADOS: G2 (HbA1c ≤ 7%) mostrou valores menores para media de iRR; STD RR; RR Tri, TINN, SD2, CD e maiores para media de FR quando comparado com G1 (HbA1c > 7%). Adicionalmente, HbA1c correlacionou-se negativamente com media iRR (r=0,28, p=0,044); STD RR (r=0,33, p=0,017); RR Tri (r=-0,35, p=0,013), SD2 (r=-0,39, p=0,004) e positivamente com media FC (r=0,28, p=0,045). Finalmente, a glicemia de jejum correlacionou-se negativamente com STD RR (r=-0,36, p=0,010); RR Tri (r=-0,36, p=0,010); TINN (r=-0,33, p=0,019) e SD2 (r=-0,42, p=0,002). CONCLUSAO: Conclui que o controle glicemico deficiente relaciona-se com indices de modulacao autonomica cardiaca em individuos com DM2, ainda que nao apresentem neuropatia autonomica cardiovascular.INTRODUCCION: Es de conocimiento general que la diabetes mellitus tipo 2 (DM2) produce neuropatia autonomica cardiovascular (NAC), que puede afectar la modulacion autonomica cardiaca. Entretanto, no es claro si la falta de control glucemico en diabeticos tipo 2 sin NAC, podria impactar negativamente en la modulacion autonomica cardiaca. Objetivo: Evaluar la relacion entre control glucemico y modulacion autonomica cardiaca en individuos con DM2 sin neuropatia autonomica cardiovascular. Estudio descriptivo, prospectivo y transversal. METODOS: Cuarenta y nueve pacientes con DM2 (51±7 anos) fueron divididos en dos grupos de acuerdo con la hemoglobina glucosilada (HbA1c): G1: ≤ 7% y G2: >7,0%. La frecuencia cardiaca de reposo (FC) e intervalo RR (iRR) fueron obtenidos y calculados por metodos lineales (promedio iRR; promedio FC; rMSSD; STD RR; LF; HF; LF/HF, TINN y RR Tri) y no lineales (SD1; SD2; DFα1; DFα2, Entropia de Shannon; ApEn; SampEn y CD) de variabilidad de frecuencia cardiaca. Fueron obtenidas insulina, HOMA-IR, glucemia en ayunas y HbA1c a traves de analisis sanguineos. RESULTADOS: G2 (HbA1c ≤ 7%) mostro valores menores para el promedio de iRR; STD RR; RR Tri, TINN, SD2, CD y mayores para el promedio de FR al ser comparado con G1 (HbA1c > 7%). Adicionalmente, HbA1c se correlaciono negativamente con el promedio iRR (r=0,28, p=0,044); STD RR (r=0,33, p=0,017); RR Tri (r=-0,35, p=0,013), SD2 (r=-0,39, p=0,004) y positivamente con el promedio FC (r=0,28, p=0,045). Finalmente, la glucemia en ayunas se correlaciono negativamente con STD RR (r=-0,36, p=0,010); RR Tri (r=-0,36, p=0,010); TINN (r=-0,33, p=0,019) e SD2 (r=-0,42, p=0,002). CONCLUSION: Concluimos que el control glucemico deficiente se relaciona con indices de modulacion autonomica cardiaca en individuos con DM2, aunque no presenten neuropatia autonomica cardiovascular.


European Journal of Physical and Rehabilitation Medicine | 2015

Resistance exercise training improves heart rate variability and muscle performance: a randomized controlled trial in coronary artery disease patients

Flávia Cristina Rossi Caruso; Ross Arena; Shane A. Phillips; José Carlos Bonjorno; Renata Gonçalves Mendes; Vivian Maria Arakelian; Daniela Bassi; C. Nogi; Audrey Borghi-Silva


Sports Medicine - Open | 2016

Potential Effects on Cardiorespiratory and Metabolic Status After a Concurrent Strength and Endurance Training Program in Diabetes Patients — a Randomized Controlled Trial

Daniela Bassi; Renata Gonçalves Mendes; Vivian Maria Arakelian; Flávia Cristina Rossi Caruso; Ramona Cabiddu; José Carlos Bonjorno Júnior; Ross Arena; Audrey Borghi-Silva

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Audrey Borghi-Silva

Federal University of São Carlos

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Renata Gonçalves Mendes

Federal University of São Carlos

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Daniela Bassi

Federal University of São Carlos

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Daniela Dutra

Federal University of São Carlos

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