William C. Levin
University of Texas Medical Branch
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Featured researches published by William C. Levin.
The American Journal of Medicine | 1966
Stephan E. Ritzmann; Earl J. Stoufflet; Elsie W. Houston; William C. Levin
M ONOCLONAL gammopathy [ 7,2], usually associated with myeloma and primary macroglobulinemia (WaldenstrGm), has also been reported in association with a variety of myeloproliferative disorders, including polycythemia [&9], myelofibrosis [3,8,9], myelofibrosis with myeloid metaplasia (31, osteomyelosclerosis [Will, erythremic myelosis [ 721, erythroleukemia [73], acute myeloblastic or lymphoblastic leukemia [7] and chronic myelocytic leukemia [74-791. Dittmar et al. have reported a biclonal gammopathy in association with polycythemia [20]. We have observed in an adult male subject the coincidence of chronic myelocytic leukemia, monoclonal gammopathy (IgG, type K) and chromosomal abnormalities characterized by several stem lines, one of which exhibited both the Phr chromosome and a chromosomal aberration heretofore observed to be associated with primary macroglobulinemia (WaldenstrBm).
The Journal of Pediatrics | 1967
Armond S. Goldman; Stephan E. Ritzmann; Elsie W. Houston; Shirley Sidwell; Raymond L. Bratcher; William C. Levin
Detailed studies including chromosomal analysis are recorded for two male children withthe dysgammaglobulinemic antibody deficiency syndrome, and previously reported cases of this disorder are reviewed.
Annals of Internal Medicine | 1964
Elsie W. Houston; William C. Levin; Stephan E. Ritzmann
Excerpt Reports in the literature of various chromosomal abnormalities associated with leukemias continue to appear (1-9). The subject of this report is a white female patient with chronic myelocyt...
The New England Journal of Medicine | 1962
Ted P. Bond; William C. Levin; D. R. Celander; M. Mason Guest
SYMPTOMATIC hemophilia in females, with clinical and laboratory data, has been rarely demonstrated, although cases of clinically apparent hemophilia in females who were assumed to be homozygous for...
Experimental Biology and Medicine | 1950
Rose G. Schneider; William C. Levin
Summary 1. Erythrocytes from 2 individuals with sickle cell anemia, when injected into rabbits, produced antisera which agglutinated erythrocytes of 19 individuals with sickle cell anemia, but not those of 21 individuals with sickle cell trait nor those of 124 normal individuals. 2. Such sera may be useful in the differential diagnosis between sickle cell anemia and sickle cell trait. 3. The presence of agglutinin antibody has been demonstrated in the sera of all of 13 patients with sickle cell anemia. This finding may explain the hemolysis which is characteristic of this disease. 4. Of 11 patients with sickle cell trait, definite agglutinin antibody was demonstrated in the sera of 4, and doubtful agglutinin antibody in 2.
Experimental Biology and Medicine | 1970
F. J. Muniz; Elsie W. Houston; L. Cruz-Abad; Stephan E. Ritzmann; William C. Levin
Summary Unstimulated and phytohemagglutinin-stimulated lymphocytes from nine human subjects were suspended in Cetrimide solution before and after tissue culture. Their nuclear volumes were measured serially with a particle counter—400 channel analyzer device. Variations of the volume distribution curves correlated well with the morphological changes associated with blastoid transformation. This method permits rapid and objective assay of lymphocyte transformation.
Experimental Biology and Medicine | 1957
C. E. Hall; William C. Levin; O. Hall
Summary Rats which have developed parabiosis intoxication show, in addition to the other hematologic aberrations which characterize the condition, changes in the blood proteins. Anemic partners have a low plasma protein level and polycythemic animals an elevated plasma protein titre. The partitions which albumin and the respective globulins contribute toward the total serum proteins average the same in both anemic and polycythemic twins, although as compared to control levels there is a diminished serum albumin and an elevated α2-globulin component in each.
JAMA Internal Medicine | 1968
William C. Levin
This is volume 3 of the second edition of an encyclopedic treatment of hemolytic disorders. It is unfortunate that volumes 1 and 2 were published seven and five years respectively before volume 3, because the earlier volumes are now incomplete, in light of subsequent developments. Volume 3 considers hemolytic anemias which are secondary or symptomatic in origin. The organization of the material is such that the monograph is presented in three chapters. Chapter 1 (13) deals with hemolytic states associated with Hodgkins disease, leukemia, and other malignancies — particularly those involving the reticuloendothelial structures. Chapter 2 (14) discusses hemolytic disease associated with lupus erythematosus, ulcerative colitis, liver and renal diseases, and the vasculitides in general. Chapter 3 (15) examines hemolytic states associated with a heterogenous group of diseases, including infections, sarcoidosis, pregnancy, hypersplenism, vitamin deficiencies, radiation, hyperoxia, marrow hypoplasia, burns, cardiac surgery, valvular disease of the heart, and march hemoglobinuria.
JAMA Internal Medicine | 1961
Stephan E. Ritzmann; William C. Levin
JAMA Internal Medicine | 1975
Stephan E. Ritzmann; Demetrius Loukas; Hideto Sakai; Jerry C. Daniels; William C. Levin