Yoshiaki Shimizu

The resistance of P. acnes–primed interferon γ–deficient mice to low-dose lipopolysaccharide-induced acute liver injury

Transfer of "infectious" cardiac allograft tolerance induced by donor-specific transfusion.

Intrathymic alloantigen-mediated, tolerant, completely major histocompatibility complex–mismatched mouse hearts are specifically rejected by adoptively transferred anti–class I Ld+–specific 2C cells

Portal venous tolerance in the anti–class I Ld alloantigen 2C transgenic mouse ☆

Donor-specific antigen transfusion-mediated cardiac allograft tolerance is prevented by prior treatment with anti-CD8, but not anti-CD4, antibody

Organ transplant specificity of tolerance to skin grafts with heart or kidney grafts plus nondepleting anti-CD4 monoclonal antibody (RIB 5/2) and intravenous donor alloantigen administration

INTERFERON-GAMMA (IFN-γ) IS REQUIRED FOR PROPIONIBACTERIUM ACNES (PA)-PRIMED LPS HEPATIC INJURY: 150

Delayed type hypersensitivity in the CD8+ dominant anti-class I Ld alloantigen 2C transgenic mouse is mediated by small but significant CD4 population ☆

Intrathymic alloantigen-mediated tolerant, completely mhc-mismatched mouse hearts are specifically rejected by adoptively transferred in vitro-sensitized anti-class I Ld+-specific 2C cells

Immunogenicity of Ld+ transgenic mouse hearts***