Amin A. Faris
University of Tennessee
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Neurology | 1963
Amin A. Faris; Charles M. Poser; Douglas W. Wilmore; Colvin H. Agnew
BECAUSE OF THE DEVELOPMENT of new technics for visualizing the arterial supply to the brlriii and the increasing interest in surgery for the removal of obstructive lesions demonstrated by such technics, we have felt that determining the true significance of such abnormalities was imperative. In order to do this, it seemed essential to obtain :ideqn;ite radiologic visualization of the neck vessels in a group of completely healthy persons, specifically, those who had never exhibited the signs and symptoms of cerelxovuscular insufficiency. We wanted to see whetlrer there was a direct causal relationship hetwwi occlusive and obstructive lesions of the ireck vessels and the manifestation of neurologic signs and symptoms. A p u p of inmates at the Kansas State Penitentiary in Lansing, Kansas, volunteered 3s subjects for the performance of transbrachial percutaneous arteriography for this purpose.
Neurology | 1972
Amin A. Faris
Disturbance of central nervous system function is a known and well-documented complication of renal failure. However, a consistent morphologic’ or physiologic’ counterpart to the clinical picture has not been clearly established. The more recently described morphologic changes noted by S ~ h n e c k ~ , ~ may be ascribed t o specific therapeutic agents used in the modern treatment of uremia. Structural and physiologic alterations affecting the peripheral nerves in uremia have been well documented.’ ¶6 We have recently encountered a patient with renal insufficiency who, a t postmortem examination, was found to have changes characteristic of Wernicke’s encephalopathy. To our knowledge, one similar case has been described by Lopez and C ~ l l i n s ; ~ this patient, in addition to findings of Wernicke’s encephalopathy, had a lesion characteristic of central pontine myelinolysis.8
Neurology | 1964
Amin A. Faris; Charles M. Poser
DUHISG the course of investigations conducted in patients suffering from cerebrovascular insufficiency as well as in a series of healthy men,’.‘ we concluded that the presence of obstructive lesions in the vessels of the neck was not in itself sufficient to explain the occurrence of neuroIogic signs and symptoms. Many previous investigators have indicated the complexity of this matter and stated their belief that a number of factors must combine in order to produce such neurologic deficits. We have postulated that 2 basic types of lesions must be considered-the intra-arterial atherosclerotic type and the extra-arterial nonatherosclerotic type. It is evident that at least 2 different mechanisms must be responsible for the production of symptomatology. In the nonatherosclerotic group, direct mechanical obstruction appears to pIay an important role, whereas, in the atherosclerotic type, the situation is vastly more complicated. Among patients with intra-arterial atherosclerotic obstructive lesions in the neck vessels, we have encountered a large number suffering from hypertensive cardiovascular disease and diabetes, such patients not infrequently receiving a variety of saluretic drugs. Our concern with the possible role of hyponatremia in the pathogenesis of the episodes of cerebrovascular insufficiency has led to the present experimental investigation.
Neurology | 1969
Oscar E. Espinas; Amin A. Faris
Case 1. C. G., a Negro boy born on November 27, 1964, was first admitted to Children’s Mercy Hospital January 24, 1966, because of poor development and growth. He was the first of a set of identical twins born spontaneously four weeks prematurely. His birth weight was 4 Ib. 3 oz. The neonatal course was uncomplicated. He was noted to be able to control his head at the age of 4 months and to sit up at 6 months. He had no known allergies and had been given all routine immunizations. The family has a strong tendency towards twinning. The mother is one of twin siblings and has a sister who also gave birth to a set of twins. At the age of 7 months the patient had difficulty in holding up his head. At about the same time he developed convergent strabismus. His appetite became poor and he started to vomit. His formula was changed to semifluid feeding, but these symptoms progressed. He also developed generalized spasticity with extreme arching of the neck and hyperextension of the extremities. On admission he was poorly nourished. His mouth could be opened only with extreme difficulty and had pooling of secretions. He had scattered rhonchi in his lungs and his abdomen was rigid and board-like, but bowel sounds were heard. The neurologic examination revealed opisthotonus and striking convergent strabismus, with no other ocular abnormalities. The infant had noticeable generalized spasticity, hyperreflexia with bilateral unsustained clonus, and plantar extensor responses. In the hospital he developed bilateral bronchopneumonia, which was treated with antibiotics. His activity was limited to rolling from side to side. His oral secretions became a problem and feeding became more difficult. Grunting respiration very similar to laryngeal stridor was noticeable. He was discharged after three weeks of hospitalization. He was brought back to the emergency room dead on arrival on April 19, 1966, at the age of 17 months. Laboratory results are shown in the table.
Neurology | 1963
Amin A. Faris; Charles M. Poser; Colvin H. Agnew; Karl Youngstrom
As A RESULT of the increasing interest in the state of the neck vessels in patients suffering from cerebrovascular diseases, various methods for the visualization of those vessels have been proposed and used. We have recently reported on our experience with a method which allows visualization of all 4 neck vessels during a single radiographic exposure.192 A few investigators have described the effects of radiopaque contrast materials upon the electroencephalogram both in experimental animals and in patients, hut to our knowledge these have not been described in brachiocephalic angiography in which large amounts of contrast material are used.
Neurology | 1972
Amin A. Faris; Helio Lemmi
The neuropathology of epilepsy in man has disclosed the finding of Ammon’s horn sclerosis in about half the patients studied. It is controversial whether the phenomenon is a primary cause of seizure disorder or is secondary to seizure activity. The published data pertinent to the question has been inconclusive.’ y 2 The purpose of this investigation was to determine whether induced seizures could result in Ammans horn sclerosis in the experimental animal. An unexpected finding of diencephalic sclerosis and no abnormality in the medial temporal lobe structures prompted this preliminary report.
JAMA | 1991
Marc R. Mayberg; S. Eric Wilson; Frank Yatsu; David G. Weiss; Louis M. Messina; Linda A. Hershey; Cindy Colling; Joseph Eskridge; Daniel Deykin; H. Richard Winn; Phillip Kistler; P.J. Mohr; Hermes Kontos; Mimi Platt; Calvin B. Ernst; Lawrence R. Wechsler; Elmer Hall; Martin Weiss; Rose Kurz; Edgard Perez; Daniel J. Safer; Rev Maurice Moore; Thomas Hobbins; Megan Arthur; Allen Raskin; Rep Martin Feldbush; Mike Lee; Diana Preston; Debbie Davis; Linda Dunford
JAMA Neurology | 1963
Amin A. Faris; Creighton A. Hardin; Charles M. Poser
JAMA | 1962
Charles M. Poser; R. Glenn Snodgrass; Amin A. Faris
JAMA Neurology | 1963
Creighton A. Hardin; Thomas H. Hendren; Amin A. Faris; Charles M. Poser