Antonio Noda

Cutaneous primary paracoccidioidomycosis.

Paracoccidioidomycosis is a common fungal infection in Latin America. Few cases have been described in non‐endemic countries and their diagnosis without the correct suspicion, bearing in mind that the latency period might be up to 60 years, may not be easy.

Multiple verrucae vulgaris in a young woman's tattoo.

356 JEADV 2006, 20, 341–362

Sweet’s Syndrome Induced by Oral Contraceptive

All criteria for drug-induced Sweet’s syndrome are fulfilled by our patient. Only a single case in which Sweet’s syndrome was believed to be precipitated by oral contraceptives could be found. Nevertheless as more cases of drug-induced Sweet’s syndrome are being reported in the literature we suggest that a drug aetiology should be sought in cases of Sweet’s syndrome and we recommend that a careful drug history should be included in the assessment of all the cases of this syndrome. (excerpt)

Primary cutaneous mucormycosis: a diagnosis to consider

Primary cutaneous mucormycosis is a deep fungal infection, mainly seen in diabetics and immunocompromised subjects. Rapid diagnosis and therapy are necessary to avoid fatal outcome. We describe the complete histopathological and microbiological studies of primary cutaneous mucormycosis in a 74‐year‐old man with several risk factors, such as chronic obstructive pulmonary disease, respiratory acidosis, hemolytic anemia, myelodysplastic syndrome and iatrogenic diabetes, due to corticosteroid therapy. He developed two cutaneous necrotic scars on his left leg. Mucormycosis was suspected and specimens from surgical debridement were histopathologically and microbiologically studied confirming the clinical diagnosis. Amphotericin B was given topically and intravenously resulting in complete healing of the ulcer. Risk factors and microbiological studies are compared with those in the current literature. It is necessary in certain cases to suspect mucormycosis infections in diabetics, immunocompromised subjects and even in healthy individuals. Rapid diagnosis and treatment are important, but they should be based on complete histopatholgical and microbiological studies, to establish the genus of the causal agent.

PUVA therapy in localized scleroderma

To the Editor: We report the case of an 80-yearold man with localized scleroderma, treated successfully with PUVA therapy. The patient came to our office with progressive hardening of his abdominal skin. Clinical examination (Fig. 1) showed a large (25 cm in diameter) slightly erythematous plaque that could not be pinched and made it difficult for the patient to bend down. The lesion margins were rough, showing no lilac ring. Histological examination confirmed morphoea. The patient was classified as skin phototype IV. PUVA therapy began with 0.6 mgk, 42 mglday 8-methoxypsoralen and an initial UVA dose of 4 J/ cm2, which was gradually increased, weekly, to a maximal single dose of 18 J/cm2. Photochemotherapy was given once daily, three times a week from March to November 1996. Two months after the patient started treatment he felt better; in fact, soft areas could be seen inside the plaque. In November he was able to move around well, and photochemother-

Atypical molluscum contagiosum.

JEADV 2006, 20 , 461–488

Unilateral naevoid telangiectasia in a young man after chemotherapy: a simple coincidence or a new clinical association?

JEADV 2006, 20 , 999–1032

Infliximab‐induced eczematoid eruption

JEADV 2006, 20, 1133–1167