J. Lowry Miller
Columbia University
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Archives of Dermatology | 1959
J. Lowry Miller
Dr. G. V. Kulchar : I thought in looking over the lesions that there was atrophy, which would not occur in the healing of a tuberculid. The condition looked to me more like that described as folliculitis ulerythematosa reticulata. Dr. H. E. Miller : It is difficult to make a positive diagnosis in this case with¬ out microscopic examination of tissue. However, I believe that the diagnosis as given is correct. Dr. A. E. Ingels : Clinically, I feel that this condition is amyloidosis or a similar degenerative disease. The lesions are semitranslucent, brownish and cyst¬ like or granular to touch, which would suggest to me a degenerative process. Naturally, the diagnosis would hinge on a biopsy with differential stains. Dr. H. V. Allington, Oakland, Calif. : I believe the disease is a rosacea-like tuberculid. I think the atrophy in this case, if any is present, is rather indistinct and poorly defined. The lesions individually correspond to those of a rosacea-like tuberculid. Dr. H. J. Templeton, Oakland, Calif.: Here on the Pacific Coast physicians have few opportunities to see Lewandowskys rosacea-like tuberculid. Therefore I am not very familiar with it. However, in my opinion, my patient has this disease. The rosacea-like eruption spares rather than involves the middle third of her face. There is an absence of any pustular element. As some of the papules disappear their sites seem to be somewhat atrophie. Dr. A. E. Ingels : Do you not feel that it is important that the patient has teen in the best of health? She never had tuberculosis, and there was never any in the family. She feels and looks perfectly well and robust.
Archives of Dermatology | 1953
Marion B. Sulzberger; J. Lowry Miller
Leprosy, Lepromatous Type: Response to Isoniazid Therapy. Presented byDr. William DirectorandDr. Roman Lysiak. This patient, W. Q. C., was admitted to the hospital on July 3, 1952, because of pea-sized and smaller infiltrated nodules on the face (especially on the nose), extremities, and buttocks. The lesions started to appear on the left cheek one year (?) before admission and gradually the process spread over the other surfaces of the skin. Treatment consisted of administration of isoniazid (Rimifon), 150 mg. daily from July 10 to Aug. 25 (in total, 7 gm. and 50 mg.) and an isopropyl derivative of isoniazid (Marsilid), 150 mg. daily from Aug. 26 to Nov. 6 (in total, 10 gm. and 650 mg.). There has been definite improvement; numerous nodules diminished in size, and some disappeared completely. Laboratory findings on admission were as follows: A blood cell count gave the following results: red blood
JAMA | 1957
J. Lowry Miller; Marvin Brodey; Justina H. Hill
JAMA | 1954
J. Lowry Miller; Meyer H. Slatkin; Marvin Brodey; Harry L. Wechsler; Justina H. Hill
Archives of Dermatology | 1949
J. Lowry Miller; Meyer H. Slatkin; Balbina A. Johnson
JAMA | 1968
William G. Atwood; J. Lowry Miller; Genevieve W. Stout; Leslie C. Norins
JAMA | 1952
J. Lowry Miller; Meyer H. Slatkin; Rose R. Feiner; Joseph Portnoy; A. Benson Cannon
Journal of Investigative Dermatology | 1948
J. Lowry Miller; Meyer H. Slatkin; Balbina A. Johnson
JAMA | 1956
J. Lowry Miller; Meyer H. Slatkin; Justina H. Hill
Archives of Dermatology | 1969
William G. Atwood; J. Lowry Miller