Lorraine Chuman
University of California, San Diego
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Publication
Featured researches published by Lorraine Chuman.
Cell | 1991
Mark Stamnes; Bih-Hwa Shieh; Lorraine Chuman; Greg L. Harris; Charles S. Zuker
Mutations in the Drosophila ninaA gene cause dramatic reductions in rhodopsin levels, leading to impaired visual function. The ninaA protein is a homolog of peptidyl-prolyl cis-trans isomerases. We find that ninaA is unique among this family of proteins in that it is an integral membrane protein, and it is expressed in a cell type-specific manner. We have used transgenic animals misexpressing different rhodopsins in the major class of photoreceptor cells to demonstrate that ninaA is required for normal function by two homologous rhodopsins, but not by a less conserved member of the Drosophila rhodopsin gene family. This demonstrates in vivo substrate specificity in a cyclophilin-like molecule. We also show that vertebrate retina contains a ninaA-related protein and that ninaA is a member of a gene family in Drosophila. These data offer insights into the in vivo role of this important family of proteins.
Journal of Cell Biology | 1979
Rindler Mj; Lorraine Chuman; Lisa G. Shaffer; Milton H. Saier
Proceedings of the National Academy of Sciences of the United States of America | 1979
Mary Taub; Lorraine Chuman; Milton H. Saier; Gordon Sato
Cancer Research | 1976
Charles D. Stiles; Walter Desmond; Lorraine Chuman; Gordon Sato; Milton H. Saier
Cancer Research | 1976
Charles D. Stiles; Walter Desmond; Lorraine Chuman; Gordon Sato; Milton H. Saier
FEBS Journal | 1989
Lorraine Chuman; Stuart Brody
Journal of Supramolecular Structure and Cellular Biochemistry | 1981
Mary Taub; Ben Ü; Lorraine Chuman; Rindler Mj; Milton H. Saier; Gordon Sato
Journal of Cellular Physiology | 1983
Mary Taub; Milton H. Saier; Lorraine Chuman; Sue Hiller
Journal of Cellular Physiology | 1985
Paula Boerner; Rindler Mj; Lorraine Chuman; Milton H. Saier
Archive | 1976
Charles D. Stiles; Walter Desmond; Lorraine Chuman; G S Saier; O C In