Mina Laghmari

Cilioretinal artery and central retinal vein occlusion after sildenafil use

Its reported that sildenafil may cause non-arteritic anterior ischaemic optic neuropathy and transient changes in perception of colour hue or brightness.1 We report a case of cilioretinal artery and central retinal vein occlusion after sildenafil use. A previously healthy 40-year-old man, presented with acute visual loss of the left eye. He took one tablet of sildenafil (100 mg) for two consecutive days before the onset of symptoms. At examination the visual acuity was …

Unilateral multifocal choroidal ischemia revealing a giant cell arteritis: a case report.

Unilateral multifocal choroidal ischemia revealing a giant cell arteritis: a case report Zouheir Hafidi, Hanan Handor, Hamid Elmoussaif, Mina Laghmari, Abdelouahed Karmane, Samira Tachfouti, Rajae Daoudi Université Mohammed V Souissi, Service d’Ophtalmologie A de l’Hôpital des Spécialités, Centre Hospitalier Universitaire, Rabat, Maroc Corresponding author: Zouheir Hafidi, Université Mohammed V Souissi, Service d’Ophtalmologie A de l’Hôpital des Spécialités, Centre Hospitalier Universitaire, Rabat, Maroc

Le syndrome d’Usher: à propos d’une observation

Usher syndrome is a genetic disease resulting in double sensory deprivation (auditory and visual) called deafblindness. We report the case of a 50-year old patient, born to consanguineous parents, presenting with congenital deafness associated with normal vestibular function and pigmentary retinopathy responsible for decreased bilateral visual acuity occurred at the age of 16 years. This association composes Usher syndrome type 2, a rare autosomal recessive disorder. Cataract surgery allowed visual acuity improvement in this patient.

Persistent hyperplastic primary vitreous (PHPV)

A 14-year-old otherwise healthy boy presented with rightsided small eye with leukocoria since the age of four. At that time, his parents neglected these signs until the leukocoria bothered the patient who asked for treatment. His visual acuity was reduced to hand motion on the right eye. External examination revealed a right esotropia with leukocoria. Slit-lamp examination of the right eye revealed small corneal diameters and a shallow anterior chamber (Fig. 1A). Furthermore, we noted a posterior synechia (Fig. 1A, white arrow), elongated and stretched ciliary processes (Fig. 1A, white star) with a white and partially resorbed cataract preventing funduscopy. The left eye was normal. Ocular B-scan and Doppler ultrasonography of the right eye showed a short axial length (19.7 mm, normal > 21 mm), a cataractous lens and an echogenic dense

Long standing well-tolerated intracorneal thorns

Figure 1. Slit lamp examination showing two intracorneal thorns (blac heads, C and D). The largest one projected in the anterior chamber (ar chamber, after removal of the largest thorn. F. Aspect after 6 months fo d a t ungal ocular infections. Herein, we present an unusual case f intracorneal wooden foreign bodies that were well tolerted for a period of one year. A 55-year-old man, presented for a routine ocular exam. n examination his visual acuity was 20/20 bilaterally. On lit lamp examination, two vegetal thorns (black arrows,

Scleral granuloma revealing intraocular foreign body

When confronted with any ocular trauma, the presence of an intraocular foreign body should be considered even in the absence of an obvious wound because of the risk of infection and metallosis.1 The presence of foreign bodies in certain localizations can be difficult to detect, in particular the anterior chamber angle and ciliary body. We report the atypical case of a child with sclera granuloma revealing a foreign body. This case involves 6-year-old child who consulted the emergency department for red …

A pencil injury to the eye.

Cranio-orbital foreign bodies are rare. Their ocular and neurological complications vary according to their nature and location. In children, the foreign body can be misdiagnosed if the trauma occurs in the absence of parental control. A 6-year-old girl was brought to the emergency department by her parents with a non-resolving painful swelling of the left orbit with total ophthalmoplegia and severe vision loss of the left eye 1 week after sustaining a self-harm orbital penetrating injury by a pencil. This resulted in an eyelid wound, which was sutured by a …

Congenital hypertrophy of the retinal pigment epithelium in Gardner's syndrome

A 20-year-old woman presented for a routine eye examination. Her best-corrected visual acuity was 20/20 in the right eye and 20/25 in the left eye. Fundus examination revealed in both eyes the presence of multiple egg-shaped hyperpigmented retinal lesions (at least 4), sur-rounded by a depigmented halo(figure 1, black arrows). The appearance of these lesions was suggestive of congenital hypertrophy of the retinal pigment epithelium (CHRPE). The patients medical history and general examination were unremarkable but her family history was pertinent for familial adenomatous polyposis (FAP) in a sister and colon cancer in her mother. The patient underwent colonoscopy and she found to have adenomatous polyps consistent with the diagnosis of Gadners syndrome (GS). She then was scheduled for bi-yearly screening colonoscopy. CHRPE are congenital hamartomas of retinal pigment epithelium. They can occur as solitary or multiple, they may be found in the normal population and are usually observed during routine ophthalmos copy. Multiple or bilateral CHRPE mayoccur in Familial Adenomatous Polyposis (FAP), an autosomal dominant diseasecaused by mutations in the adenomatous polyposis coli (APC) gene. This entity istermed Gadners syndrome which include prominent intestinal lesions and extracolonic manifestations such as osteomas, skin tumors, supernumerary teeth, desmoid tumor and CHRPE. Colorectal examination is crucial for early intervention and treatment, as the colon polyps progress to malignancy in nearly 100% of cases. Multiple and bilateral CHRPE in FAP isconsidered a clinical disease marker. However, the absence of CHRPE has no predictive value for absence of GS or FAP.