John Paul Wright

Association of prenatal and childhood blood lead concentrations with criminal arrests in early adulthood

Background Childhood lead exposure is a purported risk factor for antisocial behavior, but prior studies either relied on indirect measures of exposure or did not follow participants into adulthood to examine the relationship between lead exposure and criminal activity in young adults. The objective of this study was to determine if prenatal and childhood blood lead concentrations are associated with arrests for criminal offenses. Methods and Findings Pregnant women were recruited from four prenatal clinics in Cincinnati, Ohio if they resided in areas of the city with a high concentration of older, lead-contaminated housing. We studied 250 individuals, 19 to 24 y of age, out of 376 children who were recruited at birth between 1979 and 1984. Prenatal maternal blood lead concentrations were measured during the first or early second trimester of pregnancy. Childhood blood lead concentrations were measured on a quarterly and biannual basis through 6.5 y. Study participants were examined at an inner-city pediatric clinic and the Cincinnati Childrens Hospital Medical Center in Cincinnati, Ohio. Total arrests and arrests for offenses involving violence were collected from official Hamilton County, Ohio criminal justice records. Main outcomes were the covariate-adjusted rate ratios (RR) for total arrests and arrests for violent crimes associated with each 5 μg/dl (0.24 μmol/l) increase in blood lead concentration. Adjusted total arrest rates were greater for each 5 μg/dl (0.24 μmol/l) increase in blood lead concentration: RR = 1.40 (95% confidence interval [CI] 1.07–1.85) for prenatal blood lead, 1.07 (95% CI 0.88–1.29) for average childhood blood lead, and 1.27 (95% CI 1.03–1.57) for 6-year blood lead. Adjusted arrest rates for violent crimes were also greater for each 5 μg/dl increase in blood lead: RR = 1.34 (95% CI 0.88–2.03) for prenatal blood lead, 1.30 (95% CI 1.03–1.64) for average childhood blood lead, and 1.48 (95% CI 1.15–1.89) for 6-year blood lead. Conclusions Prenatal and postnatal blood lead concentrations are associated with higher rates of total arrests and/or arrests for offenses involving violence. This is the first prospective study to demonstrate an association between developmental exposure to lead and adult criminal behavior.

The Code of the Street: A Quantitative Assessment of Elijah Anderson's Subculture of Violence Thesis and Its Contribution to Youth Violence Research

Based on extensive field research, Elijah Anderson argues that the behavior of many youths is influenced by a street culture or “code” that prescribes violent reactions to interpersonal attacks and shows of disrespect. Although Anderson’s account has been well received by the criminological community, questions remain about the validity and generality of his findings. To address these issues, the authors review other (mostly quantitative) studies of youth violence and consider whether the findings of these studies are consistent with Anderson’s account. The authors also conduct analyses to determine whether Anderson’s observations can help to extend the existing body of quantitative research and, hence, further our understanding of youth violence. Overall, the results of this assessment suggest that Anderson’s observations provide valuable insight into the general problem of youth violence and that future research in this area would benefit from greater attention to the social processes he has served to highlight.

Evidence of Negligible Parenting Influences on Self‐Control, Delinquent Peers, and Delinquency in a Sample of Twins

Behavioral genetic findings continue to call into question the dominant role of parental influence. Utilizing a sample of twins from the National Longitudinal Study of Adolescent Health (Add Health), we assess the association between parenting behaviors and child self‐control, delinquent peer formation, and delinquency. Our results indicate that genetic influences and non‐shared environmental influences account for variation in these outcomes. We discuss these findings as they relate to theorizing about the role and function of parenting in the etiology of unique traits and behaviors.

Family social capital and delinquent involvement

Much research implicates parenting in the etiology of delinquency. We draw on Colemans [Coleman, J. S. (1990). Foundations of Social Theory. Cambridge, MA: Harvard University Press.] social capital perspective to test hypotheses that relate family “investment” in youth to the production of personal and social capital that, in turn, inhibits misbehavior. Using the well-known National Youth Survey (NYS), we find that family social capital produces the types of social and personal capital envisioned by Coleman, reduces delinquency across time, moderates the effects of misbehavior, and is associated with general positive effects across the life course. The implications associated with the social capital perspective are discussed.

Self-control as an executive function: reformulating Gottfredson and Hirschi's parental socialization thesis

According to Gottfredson and Hirschi (1990), levels of self-control are determined by parental management techniques, not by biological and genetic influences. Recent behavioral genetic and neuroscientific research challenges this view and reveals that biogenic factors are largely responsible for the development of self-control. The current article builds off this body of literature and argues that Gottfredson and Hirschis parental socialization thesis should be reformulated to recognize that self-control is just one part of a larger constellation of executive functions that are modulated by the prefrontal cortex of the brain. Using a sample of about 3,000 children, this reformulated thesis was tested by examining whether neuropsychological deficits are predictive of parental and teacher reports of the childs level of self-control. Results revealed that measures of neuropsychological deficits were associated with variability in childhood self-control. Theoretical implications of the findings are discussed.

Is Child Saving Dead? Public Support for Juvenile Rehabilitation

In recent years, the sustained criticism leveled at juvenile rehabilitation has raised the question of whether the public continues to endorse the correctional policy of saving youthful offenders. However, in a 1998 statewide survey of Tennessee residents, the respondents indicated that rehabilitation should be an integral goal of the juvenile correctional system. They also endorsed a range of community-based treatment interventions and favored early intervention programs over imprisonment as a response to crime. Taken together, these findings revealed that the publics belief in “child saving” remains firm, and that citizens do not support an exclusively punitive response to juvenile offenders.

A gene × gene interaction between DRD2 and DRD4 is associated with conduct disorder and antisocial behavior in males

BackgroundAntisocial behaviors are complex polygenic phenotypes that are due to a multifactorial arrangement of genetic polymorphisms. Little empirical research, however, has been undertaken that examines gene × gene interactions in the etiology of conduct disorder and antisocial behavior. This study examined whether adolescent conduct disorder and adult antisocial behavior were related to the dopamine D2 receptor polymorphism (DRD2) and the dopamine D4 receptor polymorphism (DRD4).MethodsA sample of 872 male participants from the National Longitudinal Study of Adolescent Health (Add Health) completed self-report questionnaires that tapped adolescent conduct disorder and adult antisocial behavior. DNA was genotyped for DRD2 and DRD4.ResultsMultivariate regression analysis revealed that neither DRD2 nor DRD4 had significant independent effects on conduct disorder or antisocial behavior. However, DRD2 interacted with DRD4 to predict variation in adolescent conduct disorder and in adult antisocial behavior.ConclusionThe results suggest that a gene × gene interaction between DRD2 and DRD4 is associated with the development of conduct disorder and adult antisocial behavior in males.

Low self-control and coworker delinquency: A research note

Abstract Since the publication of Gottfredson and Hirschi [A General Theory of Crime. Stanford, CA: Stanford Univ. Press, 1990], a large amount of research has shown a link between low self-control and delinquency. Some research has revealed that low self-control has not been able to account for the strong effects of peer delinquency on delinquency. Criminological literature has, until recently, neglected the interactional relationship between low self-control and delinquent peers in predicting delinquency. This study used a sample of employed high school seniors to assess the interaction between low self-control and coworker delinquency on occupational delinquency. Regression analyses indicated that the interaction term was a strong predictor of occupational delinquency, even after controlling for several established predictors of delinquency.

The Intersection of Genes and Neuropsychological Deficits in the Prediction of Adolescent Delinquency and Low Self-Control

Gottfredson and Hirschi’s A General Theory of Crime, Moffitt’s developmental taxonomy theory, and Caspi et al.’s Gene × Environment study are three of the most influential pieces of contemporary criminological scholarship. Even so, there has been little attempt to integrate and empirically assess these three perspectives simultaneously. This article addresses this gap in the literature by analyzing phenotypic and genotypic data from the National Longitudinal Study of Adolescent Health (Add Health). The results revealed that all three perspectives have considerable empirical support, where neuropsychological deficits interact with the MAOA genotype to predict adolescent delinquency and levels of self-control for White males. The theoretical implications of the findings are noted.

Delinquent Peer Group Formation: Evidence of a Gene X Environment Correlation

Emerging evidence suggests that variants of specific genes may influence some youths to seek out or associate with antisocial peers. Using genotypic data (N = 1,816) from the National Longitudinal Study of Adolescent Health (J. R. Udry, 1998, 2003), the authors tested this possibility. They found that the 10R allele of the dopamine transporter (DAT1) gene was associated with self-reported delinquent peer affiliation for male adolescents from high-risk environments (β range = .13-.14) despite controlling for delinquent involvement, self-control, and drug and alcohol use. The authors discuss the importance of using a biosocial framework to examine issues related to adolescent development.