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Dive into the research topics where Margaret A. Kolka is active.

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Featured researches published by Margaret A. Kolka.


Journal of Thermal Biology | 1993

Validation of a Temperature Telemetry System during Moderate and Strenuous Exercise

Margaret A. Kolka; Mark D. Quigley; Laurie A. Blanchard; Deborah A. Toyota; Lou A. Stephenson

1. 1.Esophageal temperature [Tes (18.0 ± 6.1 min)] and pill temperature [Thti (25.3 ± 9.1 min)] reached steady state faster (P < 0.05) during moderate exercise (40 min at 40% peak VO2) than rectal temperature [Tre (37.3 ± 4.6 min)] at Ta = 29°C, Tdp = 11°C. Steady-state exercise temperatures were lower for Tes = 37.18 ± 0.18°C andThti = 37.20 ± 0.32°C than Tre = 37.46 ± 0.15°C (P < 0.05). 2. 2.During moderate exercise the change in core temperature per time (slope) was greater (P < 0.05) for Tes (0.050 ± 0.013°C min−1) than Thti (0.031 ± 0.014°C min−1) and Tre (0.018 ± 0.005°C min−1. During intense exercise the change in Tes per minute was twice that for Thti and 5 times that for Tre. 3. 3.Overall, Thti tracked dynamic changes in core temperature significantly faster than Tre, although Thti did not track dynamic changes as well or as consistently as Tes. The concept of using a temperature sensor in a pill may be useful clinically, but mobility of the pill makes this temperature measurement less suitable for research than esophageal or rectal temperature measurements.


Medicine and Science in Sports and Exercise | 1982

Metabolic and thermoregulatory responses to exercise during the human menstrual cycle.

Lou A. Stephenson; Margaret A. Kolka; J. E. Wilkerson

Metabolic and thermoregulatory responses to exercise during the human menstrual cycle. Med. Sci. Sports Exercise, Vol. 14, No. 4, pp. 270-275, 1982. Six healthy adult females with presumptively normal menstrual cycles volunteered to exercise on a bicycle ergometer on five separate days (days 2, 8, 14, 20, and 26 of the menstrual cycle; day 1 = onset of menstruation). On each experimental day each subject exercise at four submaximal exercise intensities and at an exercise intensity that elicited a peak oxygen uptake. At rest and at each exercise intensity metabolic and thermoregulatory measurements were made. There were no changes at rest or at any exercise intensity relative to cycle day in absolute oxygen uptake (l . min-1). Similarly, mean peak oxygen uptake and average work time to exhaustion were not different during the various phases of the menstrual cycle. The mean core temperature (Tre) at each exercise intensity was elevated on days 14 and 20 above that observed during flow (day 2) and the follicular phase (day 8). This implied a dissociation of metabolic responses from thermoregulatory responses to exercise during the human menstrual cycle similar to that previously observed in exercising febrile males.


Journal of Thermal Biology | 1997

Use of an ingestible telemetry sensor to measure core temperature under chemical protective clothing

Margaret A. Kolka; Leslie Levine; Lou A. Stephenson

Abstract The purpose of this study was to describe core temperatures measured by esophageal temperature and a swallowed, telemetry sensor in women during exercise when wearing clothing with high thermal resistance ( R T , 0.4 m −2 · K −1 ·W −1 ). Resting esophageal temperature ( T es ) averaged 37.11 ± 0.21°C and resting pill temperature ( T pill ) averaged 37.17 ± 0.27°C. The combination of exercise (225 ± 30 W m −2 ), clothing and ambient temperature ( T a = 30°C) caused T es to increase to an average of 38.67 ± 0.28°C and T pill to increase to an average of 38.71 ± 0.33°C during the hour of treadmill walking. The two temperature measurements were compared by least squares regression techniques. For the eight individual experiments run, the regression coefficient ( r ) averaged 0.98 ± 0.01. The calibrated temperature sensor ingested in these experiments provided accurate, usable core temperature data during logistically difficult experimental conditions.


Pflügers Archiv: European Journal of Physiology | 1987

Cutaneous blood flow and local sweating after systemic atropine administration

Margaret A. Kolka; Lou A. Stephenson

AbstractLocalized cutaneous vasodilation (flush) is seen following systemic atropine administration. To verify calculated enhanced dry heat loss with actual changes in cutaneous blood flow, four men were studied in both control and atropine (0.025 mg·kg−1;im) experiments (Ta=30°C,Tdp=7°C) during moderate exercise (55%


Computers in Biology and Medicine | 2008

Thermoregulatory model to predict physiological status from ambient environment and heart rate

Miyo Yokota; Larry G. Berglund; Samuel N. Cheuvront; William R. Santee; William A. Latzka; Scott J. Montain; Margaret A. Kolka; Daniel S. Moran


Journal of Thermal Biology | 1997

INTERACTION OF MENSTRUAL CYCLE PHASE, CLOTHING RESISTANCE AND EXERCISE ON THERMOREGULATION IN WOMEN

Margaret A. Kolka; Lou A. Stephenson

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Journal of Thermal Biology | 1986

Environmental stress after atropine treatment

Margaret A. Kolka; Lou A. Stephenson; Richard R. Gonzalez


ASTM special technical publications | 1986

Efficacy of Heat Exchange by Use of a Wettable Cover over Chemical Protective Garments

Richard R. Gonzalez; John R. Breckenridge; Clement A. Levell; Margaret A. Kolka; K. B. Pandolf

O2 peak). Esophageal temperature (Tes) and arm sweating (


Journal of Thermal Biology | 1989

Depressed sweating during exercise at altitude

Margaret A. Kolka; Lou A. Stephenson; Richard R. Gonzalez


Journal of Thermal Biology | 1992

Cardiovascular and thermoregulatory responses to repeated anticholinesterase administration

Margaret A. Kolka; Lou A. Stephenson

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Lou A. Stephenson

United States Army Research Institute of Environmental Medicine

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Bruce S. Cadarette

United States Army Research Institute of Environmental Medicine

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Michael N. Sawka

United States Army Research Institute of Environmental Medicine

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Leslie Levine

United States Army Research Institute of Environmental Medicine

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Scott J. Montain

United States Army Research Institute of Environmental Medicine

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Janet E. Staab

United States Army Research Institute of Environmental Medicine

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William A. Latzka

United States Army Research Institute of Environmental Medicine

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K. B. Pandolf

United States Army Research Institute of Environmental Medicine

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