Stewart K. Lazow

Clinical ArticlesTransoral 2.0-mm miniplate fixation of mandibular fractures plus 2 weeks' maxillomandibular fixation: A prospective study☆☆☆★★★

Purpose: We conducted a study to assess the efficacy of intraoral treatment of mandibular fractures using a 2.0-mm miniplate and 2 weeks of maxillomandibular fixation (MMF). Patients and Methods: Forty-four mandible fractures in 31 patients with a mean of 15 days of MMF were included in this study. A 2.0-mm miniplate was adapted along Champys lines of ideal osteosynthesis and secured with four 8.0-mm monocortical screws. All patients were followed for at least 8 weeks after surgery. The incidences of bone or soft tissue infections, wound dehiscence, nonunion, malunion, malocclusion, plate fractures, and iatrogenic neurosensory deficits were prospectively evaluated. Results: Primary bone healing was achieved in 100% of cases. No soft or hard tissue infection, malocclusion, malunion, nonunion, dental injuries, plate fracture, or iatrogenic nerve injuries were observed. Two (4.52%) minor complications—intraoral wound dehiscences—were noted. Conclusions: The use of a single 2.0-mm miniplate adapted along Champys line of ideal osteosynthesis and stabilized with 4 monocortical screws plus 2 weeks of MMF was a viable treatment modality for mandibular fractures.

South American blastomycosis of the maxilla: report of a case

An unusual case of localized South American blastomycosis of the maxilla, occurring in the United States, is reported. Clinical presentation, diagnosis, treatment, and reconstruction are reviewed.

Metastatic ameloblastoma to the cervical lymph nodes: a case report and review of literature.

Ameloblastomas make up 1 % of all tumors found in the oral cavity. 1 They occur 80% of the time in the mandible, and 20% of the time in the maxilla.2 - 5 Ameloblastomas are benign tumors that are locally aggressive. Recurrence rates have been reported as high as 50% to 72%, depending on treatment modality. 1 Malignant ameloblastoma is defined as a histologically benign-appearing ameloblastoma with metastasis. 2 Metastasis most commonly occurs in the lungs (75% to 88% of cases) and lymph nodes (15% to 27% of cases), but is also seen in the liver, brain, bones, kidneys, and intestines. 3 It is theorized that metastatic spread occurs via 3 routes: lymphatics, hematogenous, and aspiration. 6,7 .

Cervicofacial Subcutaneous Emphysema: Case Report and Review of Literature

herapeutic decision making. Sclerosing agents, steoids, radiotherapy, lasers, cryotherapy, embolization, nd even continued observation represent reasonable pproaches to the vascular aspect of the lesion. evertheless, when feasible, surgical excision is the reatment method of choice and will also succeed in liminating the phlebolith. Our patient with the IMH opted for no treatment ecause she stated that other than a moderate cosetic asymmetry, she had no subjective problems. he did agree to return every 6 months for evaluation, ut she failed to present for further follow-up. In our patient with the VM, significant symptoms ad developed that prompted her to seek therapy. ormal masticatory function inevitably caused trauma ith associated hemorrhage. Furthermore, she beame increasingly embarrassed about her speech imediment. She was referred to an interventional radilogist who plans to embolize the lesion.

β-Thalassemia: Medical and Surgical Considerations in Managing Facial Deformities: Case Report and Review of the Literature

f -Thalassemia is an inherited genetic disorder characterized by a quantitative reduction of -chains of globin. More than 100 different genetic mutations affecting the -globin gene in chromosome 11 exist and ead to varying degrees of -chain production. In general, the heterozygous state is termed -thalassemia minor and is characterized by a silent, mildly hypochromic, and microcytic anemia. The homozygous state is known as -thalassemia major and requires regular blood transfusions and iron chelation therapy for survival. Approximately 10% of individuals with the homozygous trait have the clinical manifestations of intermediate severity to be termed -thalassemia ntermedia. The normal adult hemoglobin, HgA, is a tetramer consisting of 2 and 2 -globins. At 6 months of age, here is a decline in fetal hemoglobin, HgF, and subequent replacement by HgA. Therefore, the clinical anifestation of -thalassemia does not become apparent until 6 months of life when -chains are required for normal production of HgA. The absence or underproduction of -chains leads to unpaired -chains. The -globins by themselves are unstable and precipitate into erythroid precursors in the bone marrow, causing membrane damage and cell death. As a consequence, hypertrophy of erythroid marrow

Do Dental Infections Really Cause Central Nervous System Infections

In the post-World War I antibiotic era, the prevalence of central nervous system (CNS) infections is estimated to be 1 per 100,000 population. The literature is replete with anecdotal case reports of CNS infections of apparent dental etiology. Conversely, it is widely cited that the incidence of CNS infection of dental etiology is only in the range of 1% to 2%. We seek to answer the question if dental infections really cause CNS infections. In this article, we focus on septic cavernous sinus thrombosis and brain abscess and if it is a diagnosis of exclusion or evidence-based.